IL-17A mediates inflammatory and tissue remodelling events in early human tendinopathy
Published 2016 View Full Article
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Title
IL-17A mediates inflammatory and tissue remodelling events in early human tendinopathy
Authors
Keywords
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Journal
Scientific Reports
Volume 6, Issue 1, Pages -
Publisher
Springer Nature
Online
2016-06-06
DOI
10.1038/srep27149
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- (2013) Patricia R Taylor et al. NATURE IMMUNOLOGY
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- (2013) Seon-Yeong Lee et al. ARTHRITIS RESEARCH & THERAPY
- Impaired IL-17 Signaling Pathway Contributes to the Increased Collagen Expression in Scleroderma Fibroblasts
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- Regulation of gene expression in human tendinopathy
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- Mast Cells and Neutrophils Release IL-17 through Extracellular Trap Formation in Psoriasis
- (2011) A. M. Lin et al. JOURNAL OF IMMUNOLOGY
- Inflammation is Present in Early Human Tendinopathy
- (2010) Neal L. Millar et al. AMERICAN JOURNAL OF SPORTS MEDICINE
- Signaling of interleukin-17 family cytokines in immunity and inflammation
- (2010) Seon Hee Chang et al. CELLULAR SIGNALLING
- Outside-to-inside signaling through transmembrane tumor necrosis factor reverses pathologic interleukin-1β production and deficient apoptosis of rheumatoid arthritis monocytes
- (2009) Undine Meusch et al. ARTHRITIS AND RHEUMATISM
- Open versus Two Forms of Arthroscopic Rotator Cuff Repair
- (2009) Neal L. Millar et al. CLINICAL ORTHOPAEDICS AND RELATED RESEARCH
- Interleukin-1 and IL-23 Induce Innate IL-17 Production from γδ T Cells, Amplifying Th17 Responses and Autoimmunity
- (2009) Caroline E. Sutton et al. IMMUNITY
- Cytokines and apoptosis in supraspinatus tendinopathy
- (2009) N. L. Millar et al. JOURNAL OF BONE AND JOINT SURGERY-BRITISH VOLUME
- Increased mast cell numbers in human patellar tendinosis: correlation with symptom duration and vascular hyperplasia
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- The Basic Science of Tendinopathy
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- (2008) Neal L. Millar et al. CLINICAL ORTHOPAEDICS AND RELATED RESEARCH
- Loss of Homeostatic Tension Induces Apoptosis in Tendon Cells: An In Vitro Study
- (2008) Monika Egerbacher et al. CLINICAL ORTHOPAEDICS AND RELATED RESEARCH
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