Interaction of DPP10a with Kv4.3 channel complex results in a sustained current component of human transient outward current I to

Human Electrophysiological and Pharmacological Properties of XEN-D0101

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Adipocytes modulate the electrophysiology of atrial myocytes: implications in obesity-induced atrial fibrillation

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Augmentation of Kv4.2-encoded Currents by Accessory Dipeptidyl Peptidase 6 and 10 Subunits Reflects Selective Cell Surface Kv4.2 Protein Stabilization

(2012) Nicholas C. Foeger et al.   JOURNAL OF BIOLOGICAL CHEMISTRY

Transient outward K+current reduction prolongs action potentials and promotes afterdepolarisations: a dynamic-clamp study in human and rabbit cardiac atrial myocytes

(2012) A. J. Workman et al.   JOURNAL OF PHYSIOLOGY-LONDON

Dissection of the voltage-activated potassium outward currents in adult mouse ventricular myocytes: I to,f, I to,s, I K,slow1, I K,slow2, and I ss

(2011) Jie Liu et al.   BASIC RESEARCH IN CARDIOLOGY

Heteropoda Toxin 2 Interaction with Kv4.3 and Kv4.1 Reveals Differences in Gating Modification

(2011) C. V. DeSimone et al.   MOLECULAR PHARMACOLOGY

Discrepant electrophysiological characteristics and calcium homeostasis of left atrial anterior and posterior myocytes

(2010) Kazuyoshi Suenari et al.   BASIC RESEARCH IN CARDIOLOGY

A novel N-terminal motif of dipeptidyl peptidase-like proteins inactivates KV4.2 channels by a pore-blocking mechanism

(2010) Henry H. Jerng et al.   Channels

Impaired glycosylation blocks DPP10 cell surface expression and alters the electrophysiology of I to channel complex

(2010) Diego Cotella et al.   PFLUGERS ARCHIV-EUROPEAN JOURNAL OF PHYSIOLOGY

Pathology-specific effects of the IKur/Ito/IK,ACh blocker AVE0118 on ion channels in human chronic atrial fibrillation

(2008) T Christ et al.   BRITISH JOURNAL OF PHARMACOLOGY

Cellular bases for human atrial fibrillation

(2008) Antony J. Workman et al.   HEART RHYTHM

New Drugs Targeting the Cardiac Ultra-Rapid Delayed-Rectifier Current (IKur): Rationale, Pharmacology and Evidence for Potential Therapeutic Value

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Weighing the evidence for a ternary protein complex mediating A-type K+currents in neurons

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