Reduction of no-reflow and reperfusion injury with the synthetic 17β-aminoestrogen compound Prolame is associated with PI3K/Akt/eNOS signaling cascade

Title
Reduction of no-reflow and reperfusion injury with the synthetic 17β-aminoestrogen compound Prolame is associated with PI3K/Akt/eNOS signaling cascade
Authors
Keywords
Myocardial infarction, Reperfusion injury, No-reflow, Prolame, Cardioprotection, Nitric oxide
Journal
BASIC RESEARCH IN CARDIOLOGY
Volume 110, Issue 2, Pages -
Publisher
Springer Nature
Online
2015-01-14
DOI
10.1007/s00395-015-0464-y

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