A novel fusion of SQSTM1 and FGFR1 in a patient with acute myelomonocytic leukemia with t(5;8)(q35;p11) translocation

STAT3 Pathway Is Activated in ALK-positive Large B-cell Lymphoma Carrying SQSTM1-ALK Rearrangement and Provides a Possible Therapeutic Target

(2013) Emanuele S.G. d’Amore et al.   AMERICAN JOURNAL OF SURGICAL PATHOLOGY

Identification of a novel partner gene, TPR, fused to FGFR1 in 8p11 myeloproliferative syndrome

(2012) Feng Li et al.   GENES CHROMOSOMES & CANCER

A translocation t(2;8)(q12;p11) fuses FGFR1 to a novel partner gene, RANBP2/NUP358, in a myeloproliferative/myelodysplastic neoplasm

(2012) C Gervais et al.   LEUKEMIA

The kinase inhibitor TKI258 is active against the novel CUX1-FGFR1 fusion detected in a patient with T-lymphoblastic leukemia/lymphoma and t(7;8)(q22;p11)

(2011) B. Wasag et al.   HAEMATOLOGICA

SQSTM1-NUP214: a new gene fusion in adult T-cell acute lymphoblastic leukemia

(2010) P. Gorello et al.   HAEMATOLOGICA

Identification of a novel fusion, SQSTM1-ALK, in ALK-positive large B-cell lymphoma

(2010) K. Takeuchi et al.   HAEMATOLOGICA

8p11 myeloproliferative syndrome: a review

(2010) Courtney C. Jackson et al.   HUMAN PATHOLOGY

The selective autophagy substrate p62 activates the stress responsive transcription factor Nrf2 through inactivation of Keap1

(2010) Masaaki Komatsu et al.   NATURE CELL BIOLOGY

A Role for Ubiquitin in Selective Autophagy

(2009) Vladimir Kirkin et al.   MOLECULAR CELL