Kctd10 regulates heart morphogenesis by repressing the transcriptional activity of Tbx5a in zebrafish
Published 2014 View Full Article
- Home
- Publications
- Publication Search
- Publication Details
Title
Kctd10 regulates heart morphogenesis by repressing the transcriptional activity of Tbx5a in zebrafish
Authors
Keywords
-
Journal
Nature Communications
Volume 5, Issue 1, Pages -
Publisher
Springer Nature
Online
2014-01-16
DOI
10.1038/ncomms4153
References
Ask authors/readers for more resources
Related references
Note: Only part of the references are listed.- KCTD13 is a major driver of mirrored neuroanatomical phenotypes of the 16p11.2 copy number variant
- (2012) Christelle Golzio et al. NATURE
- TAL Effector-Nucleotide Targeter (TALE-NT) 2.0: tools for TAL effector design and target prediction
- (2012) Erin L. Doyle et al. NUCLEIC ACIDS RESEARCH
- Development and Validation of an Automated High-Throughput System for Zebrafish In Vivo Screenings
- (2012) Ainhoa Letamendia et al. PLoS One
- Mechanisms of T-box gene function in the developing heart
- (2011) F. Greulich et al. CARDIOVASCULAR RESEARCH
- MicroRNA-23 Restricts Cardiac Valve Formation by Inhibiting Has2 and Extracellular Hyaluronic Acid Production
- (2011) Anne Karine Lagendijk et al. CIRCULATION RESEARCH
- Nephronectin regulates atrioventricular canal differentiation via Bmp4-Has2 signaling in zebrafish
- (2011) C. Patra et al. DEVELOPMENT
- Viewing Hyaluronan
- (2011) Carol A. de la Motte et al. JOURNAL OF HISTOCHEMISTRY & CYTOCHEMISTRY
- Heritable gene targeting in zebrafish using customized TALENs
- (2011) Peng Huang et al. NATURE BIOTECHNOLOGY
- Cardiac response to startle stimuli in larval zebrafish: sympathetic and parasympathetic components
- (2010) Kara D. Mann et al. AMERICAN JOURNAL OF PHYSIOLOGY-REGULATORY INTEGRATIVE AND COMPARATIVE PHYSIOLOGY
- Kctd15 inhibits neural crest formation by attenuating Wnt/ -catenin signaling output
- (2010) S. Dutta et al. DEVELOPMENT
- Stat3 Directly Controls the Expression of Tbx5, Nkx2.5, and GATA4 and Is Essential for Cardiomyocyte Differentiation of P19CL6 Cells
- (2010) Marylynn Snyder et al. JOURNAL OF BIOLOGICAL CHEMISTRY
- Native GABAB receptors are heteromultimers with a family of auxiliary subunits
- (2010) Jochen Schwenk et al. NATURE
- Differential expression analysis for sequence count data
- (2010) Simon Anders et al. GENOME BIOLOGY
- TopHat: discovering splice junctions with RNA-Seq
- (2009) Cole Trapnell et al. BIOINFORMATICS
- Tbx20 Interacts With Smads to Confine Tbx2 Expression to the Atrioventricular Canal
- (2009) Reena Singh et al. CIRCULATION RESEARCH
- Signaling Pathways Controlling Second Heart Field Development
- (2009) Francesca Rochais et al. CIRCULATION RESEARCH
- Pdlim7 (LMP4) regulation of Tbx5 specifies zebrafish heart atrio-ventricular boundary and valve formation
- (2009) Troy Camarata et al. DEVELOPMENTAL BIOLOGY
- KCTD10 interacts with proliferating cell nuclear antigen and its down-regulation could inhibit cell proliferation
- (2009) Yibo Wang et al. JOURNAL OF CELLULAR BIOCHEMISTRY
- T-box 2, a mediator of Bmp-Smad signaling, induced hyaluronan synthase 2 and Tgf 2 expression and endocardial cushion formation
- (2009) M. Shirai et al. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA
- Reversing Blood Flows Act through klf2a to Ensure Normal Valvulogenesis in the Developing Heart
- (2009) Julien Vermot et al. PLOS BIOLOGY
- Rotation and Asymmetric Development of the Zebrafish Heart Requires Directed Migration of Cardiac Progenitor Cells
- (2008) Kelly A. Smith et al. DEVELOPMENTAL CELL
- KCTD5, a putative substrate adaptor for cullin3 ubiquitin ligases
- (2008) Yolanda Bayón et al. FEBS Journal
- Foxn4 directly regulates tbx2b expression and atrioventricular canal formation
- (2008) N. C. Chi et al. GENES & DEVELOPMENT
Discover Peeref hubs
Discuss science. Find collaborators. Network.
Join a conversationPublish scientific posters with Peeref
Peeref publishes scientific posters from all research disciplines. Our Diamond Open Access policy means free access to content and no publication fees for authors.
Learn More