Analysis of BH3-only proteins upregulated in response to oxygen/glucose deprivation in cortical neurons identifies Bmf but not Noxa as potential mediator of neuronal injury
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Title
Analysis of BH3-only proteins upregulated in response to oxygen/glucose deprivation in cortical neurons identifies Bmf but not Noxa as potential mediator of neuronal injury
Authors
Keywords
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Journal
Cell Death & Disease
Volume 5, Issue 10, Pages e1456-e1456
Publisher
Springer Nature
Online
2014-10-09
DOI
10.1038/cddis.2014.426
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- (2010) F Grespi et al. CELL DEATH AND DIFFERENTIATION
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- (2010) Seán M. Kilbride et al. JOURNAL OF BIOLOGICAL CHEMISTRY
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- (2010) Caoimhín G. Concannon et al. JOURNAL OF CELL BIOLOGY
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- Effects of transient focal cerebral ischemia in mice deficient in puma
- (2009) Katsura Kuroki et al. NEUROSCIENCE LETTERS
- Noxa: at the tip of the balance between life and death
- (2009) C Ploner et al. ONCOGENE
- In several cell types tumour suppressor p53 induces apoptosis largely via Puma but Noxa can contribute
- (2008) E M Michalak et al. CELL DEATH AND DIFFERENTIATION
- Loss of the BH3-only protein Bmf impairs B cell homeostasis and accelerates γ irradiation–induced thymic lymphoma development
- (2008) Verena Labi et al. JOURNAL OF EXPERIMENTAL MEDICINE
- The BCL-2 protein family: opposing activities that mediate cell death
- (2007) Richard J. Youle et al. NATURE REVIEWS MOLECULAR CELL BIOLOGY
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