ROS-PIASγ cross talk channelizes ATM signaling from resistance to apoptosis during chemosensitization of resistant tumors
Published 2014 View Full Article
- Home
- Publications
- Publication Search
- Publication Details
Title
ROS-PIASγ cross talk channelizes ATM signaling from resistance to apoptosis during chemosensitization of resistant tumors
Authors
Keywords
-
Journal
Cell Death & Disease
Volume 5, Issue 1, Pages e1021-e1021
Publisher
Springer Nature
Online
2014-01-24
DOI
10.1038/cddis.2013.534
References
Ask authors/readers for more resources
Related references
Note: Only part of the references are listed.- Renal Cell Carcinoma Tumors Induce T Cell Apoptosis through Receptor-Dependent and Receptor-Independent Pathways
- (2014) T. Das et al. JOURNAL OF IMMUNOLOGY
- Enhancement of radiosensitivity by a unique novel NF-κB inhibitor, DHMEQ, in prostate cancer
- (2012) N Kozakai et al. BRITISH JOURNAL OF CANCER
- The influence of reactive oxygen species on cell cycle progression in mammalian cells
- (2012) Eline Hendrike Verbon et al. GENE
- Outgrowth of Drug-Resistant Carcinomas Expressing Markers of Tumor Aggression after Long-term T RI/II Kinase Inhibition with LY2109761
- (2011) E. C. Connolly et al. CANCER RESEARCH
- ATM kinase is a master switch for the ΔNp63α phosphorylation/degradation in human head and neck squamous cell carcinoma cells upon DNA damage
- (2011) Yiping Huang et al. CELL CYCLE
- Curcumin Enhances the Efficacy of Chemotherapy by Tailoring p65NFκB-p300 Cross-talk in Favor of p53-p300 in Breast Cancer
- (2011) Gouri Sankar Sen et al. JOURNAL OF BIOLOGICAL CHEMISTRY
- NF- B Is Required for Smac Mimetic-Mediated Sensitization of Glioblastoma Cells for -Irradiation-Induced Apoptosis
- (2011) R. Berger et al. MOLECULAR CANCER THERAPEUTICS
- Sorafenib induces preferential apoptotic killing of a drug- and radio-resistant hep G2 cells through a mitochondria-dependent oxidative stress mechanism
- (2010) Jeng-Fong Chiou et al. CANCER BIOLOGY & THERAPY
- Zinc-finger transcription factor slug contributes to the survival advantage of chronic myeloid leukemia cells
- (2010) Manuela Mancini et al. CELLULAR SIGNALLING
- Gain of Cellular Adaptation Due to Prolonged p53 Impairment Leads to Functional Switchover from p53 to p73 during DNA Damage in Acute Myeloid Leukemia Cells
- (2010) Juni Chakraborty et al. JOURNAL OF BIOLOGICAL CHEMISTRY
- ATM signals to TSC2 in the cytoplasm to regulate mTORC1 in response to ROS
- (2010) A. Alexander et al. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA
- Theaflavins retard human breast cancer cell migration by inhibiting NF-κB via p53-ROS cross-talk
- (2009) Arghya Adhikary et al. FEBS LETTERS
- The combined status of ATM and p53 link tumor development with therapeutic response
- (2009) H. Jiang et al. GENES & DEVELOPMENT
- SUMOylation of Tissue Transglutaminase as Link between Oxidative Stress and Inflammation
- (2009) A. Luciani et al. JOURNAL OF IMMUNOLOGY
- A Nuclear Poly(ADP-Ribose)-Dependent Signalosome Confers DNA Damage-Induced IκB Kinase Activation
- (2009) Michael Stilmann et al. MOLECULAR CELL
- Phosphorylation of ATM by Cdk5 mediates DNA damage signalling and regulates neuronal death
- (2009) Bo Tian et al. NATURE CELL BIOLOGY
- Vaccines for the Treatment of Non-Small Cell Lung Cancer: A Renewed Anticancer Strategy
- (2009) C. Gridelli et al. ONCOLOGIST
- Snail and Slug Mediate Radioresistance and Chemoresistance by Antagonizing p53-Mediated Apoptosis and Acquiring a Stem-Like Phenotype in Ovarian Cancer Cells
- (2009) Nawneet K. Kurrey et al. STEM CELLS
- Oxidative Stress Induces Parallel Autophagy and Mitochondria Dysfunction in Human Glioma U251 Cells
- (2009) Hongyu Zhang et al. TOXICOLOGICAL SCIENCES
- GM1 and Tumor Necrosis Factor- , Overexpressed in Renal Cell Carcinoma, Synergize to Induce T-Cell Apoptosis
- (2008) T. Das et al. CANCER RESEARCH
Discover Peeref hubs
Discuss science. Find collaborators. Network.
Join a conversationPublish scientific posters with Peeref
Peeref publishes scientific posters from all research disciplines. Our Diamond Open Access policy means free access to content and no publication fees for authors.
Learn More