Influenza A virus nucleoprotein induces apoptosis in human airway epithelial cells: implications of a novel interaction between nucleoprotein and host protein Clusterin
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Title
Influenza A virus nucleoprotein induces apoptosis in human airway epithelial cells: implications of a novel interaction between nucleoprotein and host protein Clusterin
Authors
Keywords
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Journal
Cell Death & Disease
Volume 4, Issue 3, Pages e562-e562
Publisher
Springer Nature
Online
2013-03-28
DOI
10.1038/cddis.2013.89
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Note: Only part of the references are listed.- Influenza A Virus Neuraminidase Protein Enhances Cell Survival through Interaction with Carcinoembryonic Antigen-related Cell Adhesion Molecule 6 (CEACAM6) Protein
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- The Influenza A Virus NS1 Protein Interacts with the Nucleoprotein of Viral Ribonucleoprotein Complexes
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- Influenza A Virus Nucleoprotein Exploits Hsp40 to Inhibit PKR Activation
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- NP Body Domain and PB2 Contribute to Increased Virulence of H5N1 Highly Pathogenic Avian Influenza Viruses in Chickens
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- Transforming Growth Factor-β: Activation by Neuraminidase and Role in Highly Pathogenic H5N1 Influenza Pathogenesis
- (2010) Christina M. Carlson et al. PLoS Pathogens
- Autophagy, Apoptosis, and the Influenza Virus M2 Protein
- (2009) Jeremy S. Rossman et al. Cell Host & Microbe
- Hepatitis delta virus epigenetically enhances clusterin expression via histone acetylation in human hepatocellular carcinoma cells
- (2009) F.-T. Liao et al. JOURNAL OF GENERAL VIROLOGY
- Lack of Bax Prevents Influenza A Virus-Induced Apoptosis and Causes Diminished Viral Replication
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- Pathology, Molecular Biology, and Pathogenesis of Avian Influenza A (H5N1) Infection in Humans
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- Clusterin Activates Survival through the Phosphatidylinositol 3-Kinase/Akt Pathway
- (2008) Hayet Ammar et al. JOURNAL OF BIOLOGICAL CHEMISTRY
- Interaction of Polymerase Subunit PB2 and NP with Importin α1 Is a Determinant of Host Range of Influenza A Virus
- (2008) Gülsah Gabriel et al. PLoS Pathogens
- Viral Control of Mitochondrial Apoptosis
- (2008) Lorenzo Galluzzi et al. PLoS Pathogens
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