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A Novel and Potentially Unifying Mechanism for Shock Induced Early Coagulopathy

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A High Admission Syndecan-1 Level, A Marker of Endothelial Glycocalyx Degradation, Is Associated With Inflammation, Protein C Depletion, Fibrinolysis, and Increased Mortality in Trauma Patients

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Modulation of Syndecan-1 Shedding after Hemorrhagic Shock and Resuscitation

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Hemostatic Effects of Fresh Frozen Plasma May be Maximal at Red Cell Ratios of 1:2

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Therapeutic strategies targeting the endothelial glycocalyx: acute deficits, but great potential

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Circulating mitochondrial DAMPs cause inflammatory responses to injury

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The acute coagulopathy of trauma shock: Clinical relevance

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Angiopoietin-2, Marker and Mediator of Endothelial Activation With Prognostic Significance Early After Trauma?

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Extracellular histones are major mediators of death in sepsis

(2009) Jun Xu et al.   NATURE MEDICINE

Control of vascular morphogenesis and homeostasis through the angiopoietin–Tie system

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INCREASE IN ACTIVATED PROTEIN C MEDIATES ACUTE TRAUMATIC COAGULOPATHY IN MICE

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Thrombelastography and tromboelastometry in assessing coagulopathy in trauma

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The Coagulopathy of Trauma: A Review of Mechanisms

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