IL-22-producing Th22 cells play a protective role in CVB3-induced chronic myocarditis and dilated cardiomyopathy by inhibiting myocardial fibrosis
Published 2014 View Full Article
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Title
IL-22-producing Th22 cells play a protective role in CVB3-induced chronic myocarditis and dilated cardiomyopathy by inhibiting myocardial fibrosis
Authors
Keywords
Th22 cells, IL-22, Myocardial fibrosis, Chronic myocarditis, Dilated cardiomyopathy
Journal
Virology Journal
Volume 11, Issue 1, Pages -
Publisher
Springer Nature
Online
2014-12-30
DOI
10.1186/s12985-014-0230-z
References
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- (2013) Helen Benham et al. ARTHRITIS RESEARCH & THERAPY
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- (2012) C J Kim et al. Mucosal Immunology
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- (2012) Qing Kong et al. Virology Journal
- Astragaloside IV attenuates myocardial fibrosis by inhibiting TGF-β1 signaling in coxsackievirus B3-induced cardiomyopathy
- (2011) Ping Chen et al. EUROPEAN JOURNAL OF PHARMACOLOGY
- Interleukin-17A Is Dispensable for Myocarditis but Essential for the Progression to Dilated Cardiomyopathy
- (2010) G. Christian Baldeviano et al. CIRCULATION RESEARCH
- Biology of interleukin-22
- (2010) Kerstin Wolk et al. Seminars in Immunopathology
- Mechanisms of Cardiac Fibrosis in Inflammatory Heart Disease
- (2010) Gabriela Kania et al. TRENDS IN CARDIOVASCULAR MEDICINE
- Identification of a human helper T cell population that has abundant production of interleukin 22 and is distinct from TH-17, TH1 and TH2 cells
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- Production of interleukin 22 but not interleukin 17 by a subset of human skin-homing memory T cells
- (2009) Thomas Duhen et al. NATURE IMMUNOLOGY
- Ablation of Matrix Metalloproteinase-9 Increases Severity of Viral Myocarditis in Mice
- (2008) Caroline Cheung et al. CIRCULATION
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