Axl as a downstream effector of TGF-β1 via PI3K/Akt-PAK1 signaling pathway promotes tumor invasion and chemoresistance in breast carcinoma
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Title
Axl as a downstream effector of TGF-β1 via PI3K/Akt-PAK1 signaling pathway promotes tumor invasion and chemoresistance in breast carcinoma
Authors
Keywords
Axl, TGF-β1, PI3K/Akt-PAK1, Breast cancer, Invasion, Chemosensitivity
Journal
TUMOR BIOLOGY
Volume 36, Issue 2, Pages 1115-1127
Publisher
Springer Nature
Online
2014-10-20
DOI
10.1007/s13277-014-2677-3
References
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- Increased secretion of Gas6 by smooth muscle cells in human atherosclerotic carotid plaques
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- The TGF-β/Smad pathway induces breast cancer cell invasion through the up-regulation of matrix metalloproteinase 2 and 9 in a spheroid invasion model system
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- Overexpression of receptor tyrosine kinase Axl promotes tumor cell invasion and survival in pancreatic ductal adenocarcinoma
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- AXL receptor kinase is a mediator of YAP-dependent oncogenic functions in hepatocellular carcinoma
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- TGF-β1–induced expression of human Mdm2 correlates with late-stage metastatic breast cancer
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- TGF-β1 increases motility and αvβ3 integrin up-regulation via PI3K, Akt and NF-κB-dependent pathway in human chondrosarcoma cells
- (2007) Ying-Yi Yeh et al. BIOCHEMICAL PHARMACOLOGY
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