Gli-1 is crucial for hypoxia-induced epithelial-mesenchymal transition and invasion of breast cancer
Published 2014 View Full Article
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Title
Gli-1 is crucial for hypoxia-induced epithelial-mesenchymal transition and invasion of breast cancer
Authors
Keywords
Breast carcinoma, Invasion, Tumor, Epithelial-mesenchymal transition, Hypoxia, Gli-1
Journal
TUMOR BIOLOGY
Volume 36, Issue 4, Pages 3119-3126
Publisher
Springer Nature
Online
2014-12-14
DOI
10.1007/s13277-014-2948-z
References
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Related references
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- Nuclear Factor-κB–Dependent Epithelial to Mesenchymal Transition Induced by HIF-1α Activation in Pancreatic Cancer Cells under Hypoxic Conditions
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- Activation of the sonic hedgehog signaling controls human pulmonary arterial smooth muscle cell proliferation in response to hypoxia
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- Expression of the glioma-associated oncogene homolog (GLI) 1in human breast cancer is associated with unfavourable overall survival
- (2009) Anette ten Haaf et al. BMC CANCER
- Hypoxia potentiates Notch signaling in breast cancer leading to decreased E-cadherin expression and increased cell migration and invasion
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- Epithelial-Mesenchymal Transitions in Development and Disease
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- Analyzing real-time PCR data by the comparative CT method
- (2008) Thomas D Schmittgen et al. Nature Protocols
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