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Title
Suppression of Autophagic Flux by Bile Acids in Hepatocytes
Authors
Keywords
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Journal
TOXICOLOGICAL SCIENCES
Volume 137, Issue 2, Pages 478-490
Publisher
Oxford University Press (OUP)
Online
2013-11-05
DOI
10.1093/toxsci/kft246
References
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Related references
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- Tissue Specific Induction of p62/Sqstm1 by Farnesoid X Receptor
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- Liver-Specific Loss of Atg5 Causes Persistent Activation of Nrf2 and Protects Against Acetaminophen-Induced Liver Injury
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- Bile Acids Induce Inflammatory Genes in Hepatocytes
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- Dissecting the dynamic turnover of GFP-LC3 in the autolysosome
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- Involvement of members of the Rab family and related small GTPases in autophagosome formation and maturation
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- Autophagy-deficient mice develop multiple liver tumors
- (2011) A. Takamura et al. GENES & DEVELOPMENT
- Persistent activation of Nrf2 through p62 in hepatocellular carcinoma cells
- (2011) Yoshihiro Inami et al. JOURNAL OF CELL BIOLOGY
- Effect of bile duct ligation on bile acid composition in mouse serum and liver
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- Distinct Autophagosomal-Lysosomal Fusion Mechanism Revealed by Thapsigargin-Induced Autophagy Arrest
- (2011) Ian G. Ganley et al. MOLECULAR CELL
- Methods in Mammalian Autophagy Research
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- Autophagy Reduces Acute Ethanol-Induced Hepatotoxicity and Steatosis in Mice
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- Combinational Soluble N-Ethylmaleimide-sensitive Factor Attachment Protein Receptor Proteins VAMP8 and Vti1b Mediate Fusion of Antimicrobial and Canonical Autophagosomes with Lysosomes
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- Oncogenic transformation confers a selective susceptibility to the combined suppression of the proteasome and autophagy
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- A Role for NBR1 in Autophagosomal Degradation of Ubiquitinated Substrates
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- Multiple Cyclin Kinase Inhibitors Promote Bile Acid-induced Apoptosis and Autophagy in Primary Hepatocytes via p53-CD95-dependent Signaling
- (2008) Guo Zhang et al. JOURNAL OF BIOLOGICAL CHEMISTRY
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