Alk2 Regulates Early Chondrogenic Fate in Fibrodysplasia Ossificans Progressiva Heterotopic Endochondral Ossification
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Title
Alk2 Regulates Early Chondrogenic Fate in Fibrodysplasia Ossificans Progressiva Heterotopic Endochondral Ossification
Authors
Keywords
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Journal
STEM CELLS
Volume 32, Issue 5, Pages 1289-1300
Publisher
Wiley
Online
2014-01-22
DOI
10.1002/stem.1633
References
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- (2010) Damian Medici et al. NATURE MEDICINE
- Inherited human diseases of heterotopic bone formation
- (2010) Eileen M. Shore et al. Nature Reviews Rheumatology
- Recent advances in BMP receptor signaling
- (2009) Christina Sieber et al. CYTOKINE & GROWTH FACTOR REVIEWS
- BMP canonical Smad signaling through Smad1 and Smad5 is required for endochondral bone formation
- (2009) K. N. Retting et al. DEVELOPMENT
- Identification of Progenitor Cells That Contribute to Heterotopic Skeletogenesis
- (2009) Vitali Y Lounev et al. JOURNAL OF BONE AND JOINT SURGERY-AMERICAN VOLUME
- Bone morphogenetic protein heterodimers assemble heteromeric type I receptor complexes to pattern the dorsoventral axis
- (2009) Shawn C. Little et al. NATURE CELL BIOLOGY
- Classic and atypical fibrodysplasia ossificans progressiva (FOP) phenotypes are caused by mutations in the bone morphogenetic protein (BMP) type I receptor ACVR1
- (2008) Frederick S. Kaplan et al. HUMAN MUTATION
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- Early Tissue Patterning Recreated by Mouse Embryonic Fibroblasts in a Three-Dimensional Environment
- (2008) Lluís Quintana et al. TISSUE ENGINEERING PART A
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