Silencing of Fas, FADD (Fas-Associated Via Death Domain) or Caspase-3 Differentially Affects Lung Inflammation, Apoptosis and Development of Trauma Induced Septic Acute Lung Injury (ALI)

Human models of acute lung injury

(2011) A. G. Proudfoot et al.   Disease Models & Mechanisms

Caspase-8 activity has an essential role in CD95/Fas-mediated MAPK activation

(2011) A M M Kober et al.   Cell Death & Disease

Pathogenesis of indirect (secondary) acute lung injury

(2011) Mario Perl et al.   Expert Review of Respiratory Medicine

Therapeutic accessibility of caspase-mediated cell death as a key pathomechanism in indirect acute lung injury*

(2010) Mario Perl et al.   CRITICAL CARE MEDICINE

Blunt chest trauma induces mediator-dependent monocyte migration to the lung

(2010) Daniel H. Seitz et al.   CRITICAL CARE MEDICINE

Silencing of Fas-associated Death Domain Protects Mice from Septic Lung Inflammation and Apoptosis

(2009) Naoyuki Matsuda et al.   AMERICAN JOURNAL OF RESPIRATORY AND CRITICAL CARE MEDICINE

Acute Lung Injury:Apoptosis and Signaling Mechanisms

(2009) Mani Chopra et al.   EXPERIMENTAL BIOLOGY AND MEDICINE

Unknown

(2008) Mario Perl et al.   MOLECULAR MEDICINE

Evaluation of bronchoalveolar lavage fluid from ARDS patients with regard to apoptosis

(2007) Keu Sung Lee et al.   RESPIRATORY MEDICINE