Paeonol Attenuates Microglia-Mediated Inflammation and Oxidative Stress–Induced Neurotoxicity in Rat Primary Microglia and Cortical Neurons

Neuroinflammation, Oxidative Stress and the Pathogenesis of Alzheimers Disease

(2012) Paula Agostinho et al.   CURRENT PHARMACEUTICAL DESIGN

Neuroinflammation is a key player in Parkinson’s disease and a prime target for therapy

(2010) Li Qian et al.   JOURNAL OF NEURAL TRANSMISSION

Potent Anti-Inflammatory Effects of Denbinobin Mediated by Dual Inhibition of Expression of Inducible No Synthase and Cyclooxygenase 2

(2010) Hsiang-Erh Liu et al.   SHOCK

Vascular dilation by paeonol — A mechanism study

(2010) Ya-juan Li et al.   VASCULAR PHARMACOLOGY

Inhibition of LPS-Induced iNOS, COX-2 and Inflammatory Mediator Expression by Paeonol through the MAPKs Inactivation in RAW 264.7 Cells

(2009) Hee-Sung Chae et al.   AMERICAN JOURNAL OF CHINESE MEDICINE

Oxidative stress and mitochondrial dysfunction as determinants of ischemic neuronal death and survival

(2009) Kuniyasu Niizuma et al.   JOURNAL OF NEUROCHEMISTRY

Antiallergic effect of the root of Paeonia lactiflora and its constituents paeoniflorin and paeonol

(2008) Bomi Lee et al.   ARCHIVES OF PHARMACAL RESEARCH

Cyclooxygenase-1 and -2 in the Different Stages of Alzheimers Disease Pathology

(2008) J. Hoozemans et al.   CURRENT PHARMACEUTICAL DESIGN

Anti-tumor effects of paeonol in a HepA-hepatoma bearing mouse model via induction of tumor cell apoptosis and stimulation of IL-2 and TNF-α production

(2008) Guo-Ping Sun et al.   EUROPEAN JOURNAL OF PHARMACOLOGY

Paeonol inhibits RANKL-induced osteoclastogenesis by inhibiting ERK, p38 and NF-κB pathway

(2008) Huei-Yann Tsai et al.   EUROPEAN JOURNAL OF PHARMACOLOGY

Paeonol attenuates neurotoxicity and ameliorates cognitive impairment induced by d-galactose in ICR mice

(2008) Shu-Zhi Zhong et al.   JOURNAL OF THE NEUROLOGICAL SCIENCES

Protective effects of paeonol on cultured rat hippocampal neurons against oxygen–glucose deprivation-induced injury

(2007) Ji-biao Wu et al.   JOURNAL OF THE NEUROLOGICAL SCIENCES