A reactive oxygen species activation mechanism contributes to JS-K-induced apoptosis in human bladder cancer cells
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Title
A reactive oxygen species activation mechanism contributes to JS-K-induced apoptosis in human bladder cancer cells
Authors
Keywords
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Journal
Scientific Reports
Volume 5, Issue 1, Pages -
Publisher
Springer Nature
Online
2015-10-13
DOI
10.1038/srep15104
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- JS-K, a Glutathione S-Transferase–Activated Nitric Oxide Donor With Antineoplastic Activity in Malignant Gliomas
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- Oxygen toxicity: chemistry and biology of reactive oxygen species
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- Apoptosis and autophagy: Regulation of caspase-9 by phosphorylation
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- Heme Regulatory Motifs in Heme Oxygenase-2 Form a Thiol/Disulfide Redox Switch That Responds to the Cellular Redox State
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- siRNA-mediated Knockdown of the Heme Synthesis and Degradation Pathways: Modulation of Treatment Effect of 5-Aminolevulinic Acid-based Photodynamic Therapy in Urothelial Cancer Cell Lines
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- Phase I clinical trial of i.v. ascorbic acid in advanced malignancy
- (2008) L. J. Hoffer et al. ANNALS OF ONCOLOGY
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