Inhibition of the Cardiomyocyte-Specific Kinase TNNI3K Limits Oxidative Stress, Injury, and Adverse Remodeling in the Ischemic Heart
Published 2013 View Full Article
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Title
Inhibition of the Cardiomyocyte-Specific Kinase TNNI3K Limits Oxidative Stress, Injury, and Adverse Remodeling in the Ischemic Heart
Authors
Keywords
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Journal
Science Translational Medicine
Volume 5, Issue 207, Pages 207ra141-207ra141
Publisher
American Association for the Advancement of Science (AAAS)
Online
2013-10-17
DOI
10.1126/scitranslmed.3006479
References
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- Tnni3k Modifies Disease Progression in Murine Models of Cardiomyopathy
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- Overexpression of TNNI3K, a cardiac-specific MAP kinase, promotes P19CL6-derived cardiac myogenesis and prevents myocardial infarction-induced injury
- (2008) Zhong-Fang Lai et al. AMERICAN JOURNAL OF PHYSIOLOGY-HEART AND CIRCULATORY PHYSIOLOGY
- Mechanisms Underlying Acute Protection From Cardiac Ischemia-Reperfusion Injury
- (2008) Elizabeth Murphy et al. PHYSIOLOGICAL REVIEWS
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