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Expression Profiling of Nuclear Receptors Identifies Key Roles of NR4A Subfamily in Uterine Fibroids

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Gene Expression in Uterine Leiomyoma from Tumors Likely to Be Growing (from Black Women over 35) and Tumors Likely to Be Non-Growing (from White Women over 35)

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A Retinoic Acid--Rich Tumor Microenvironment Provides Clonal Survival Cues for Tumor-Specific CD8+ T Cells

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MED12 Controls the Response to Multiple Cancer Drugs through Regulation of TGF-β Receptor Signaling

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Etiology, Diagnosis, and Management of Uterine Leiomyomas

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Ulipristal Acetate versus Placebo for Fibroid Treatment before Surgery

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MED12, the Mediator Complex Subunit 12 Gene, Is Mutated at High Frequency in Uterine Leiomyomas

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Between Death and Survival: Retinoic Acid in Regulation of Apoptosis

(2010) Noa Noy   Annual Review of Nutrition

Human uterine leiomyoma-derived fibroblasts stimulate uterine leiomyoma cell proliferation and collagen type I production, and activate RTKs and TGF beta receptor signaling in coculture

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Constitutive Activation of Beta-Catenin in Uterine Stroma and Smooth Muscle Leads to the Development of Mesenchymal Tumors in Mice1

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The Fibroid Growth Study: Determinants of Therapeutic Intervention

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Cell-Specific Interaction of Retinoic Acid Receptors with Target Genes in Mouse Embryonic Fibroblasts and Embryonic Stem Cells

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Prostaglandin treatment is associated with a withdrawal of progesterone and androgen at the receptor level in the uterine cervix

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Retinoic acid treatment of human leiomyoma cells transformed the cell phenotype to one strongly resembling myometrial cells

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Growth of uterine leiomyomata among premenopausal black and white women

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