Androgen-induced PSA expression requires not only activation of AR but also endogenous IGF-I or IGF-I/PI3K/Akt signaling in human prostate cancer epithelial cells

Regulation of proliferation and differentiation of prostatic stromal cells by oestradiol through prostatic epithelial cells in a paracrine manner

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Dehydroepiandrosterone Administration or Gαq Overexpression Induces β-Catenin/T-Cell Factor Signaling and Growth via Increasing Association of Estrogen Receptor-β/Dishevelled2 in Androgen-Independent Prostate Cancer Cells

(2010) Xunxian Liu et al.   ENDOCRINOLOGY

Endocrine-Immune-Paracrine Interactions in Prostate Cells as Targeted by Phytomedicines

(2009) N. E. Gray et al.   Cancer Prevention Research

Lycopene inhibits IGF-I signal transduction and growth in normal prostate epithelial cells by decreasing DHT-modulated IGF-I production in co-cultured reactive stromal cells

(2008) Xunxian Liu et al.   CARCINOGENESIS

Androgen receptor and growth factor signaling cross-talk in prostate cancer cells

(2008) M.-L. Zhu et al.   ENDOCRINE-RELATED CANCER

Human prostate stromal cells stimulate increased PSA production in DHEA-treated prostate cancer epithelial cells

(2008) Julia T. Arnold et al.   JOURNAL OF STEROID BIOCHEMISTRY AND MOLECULAR BIOLOGY