Autophagosomes induced by a bacterial Beclin 1 binding protein facilitate obligatory intracellular infection
Published 2012 View Full Article
- Home
- Publications
- Publication Search
- Publication Details
Title
Autophagosomes induced by a bacterial Beclin 1 binding protein facilitate obligatory intracellular infection
Authors
Keywords
-
Journal
PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA
Volume 109, Issue 51, Pages 20800-20807
Publisher
Proceedings of the National Academy of Sciences
Online
2012-11-29
DOI
10.1073/pnas.1218674109
References
Ask authors/readers for more resources
Related references
Note: Only part of the references are listed.- Amino Acid Starvation Induced by Invasive Bacterial Pathogens Triggers an Innate Host Defense Program
- (2012) Ivan Tattoli et al. Cell Host & Microbe
- Selective Subversion of Autophagy Complexes Facilitates Completion of the Brucella Intracellular Cycle
- (2012) Tregei Starr et al. Cell Host & Microbe
- Autophagy: An Emerging Immunological Paradigm
- (2012) V. Deretic JOURNAL OF IMMUNOLOGY
- The Role of Atg Proteins in Autophagosome Formation
- (2011) Noboru Mizushima et al. Annual Review of Cell and Developmental Biology
- Mechanisms of Obligatory Intracellular Infection with Anaplasma phagocytophilum
- (2011) Y. Rikihisa CLINICAL MICROBIOLOGY REVIEWS
- Autophagy in immunity and inflammation
- (2011) Beth Levine et al. NATURE
- Nanoscale distribution of mitochondrial import receptor Tom20 is adjusted to cellular conditions and exhibits an inner-cellular gradient
- (2011) C. A. Wurm et al. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA
- Autophagosome targeting and membrane curvature sensing by Barkor/Atg14(L)
- (2011) W. Fan et al. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA
- Host Proteasomal Degradation Generates Amino Acids Essential for Intracellular Bacterial Growth
- (2011) C. T. D. Price et al. SCIENCE
- Mitochondria Supply Membranes for Autophagosome Biogenesis during Starvation
- (2010) Dale W. Hailey et al. CELL
- Microreview: Type IV secretion in the obligatory intracellular bacterium Anaplasma phagocytophilum
- (2010) Yasuko Rikihisa et al. CELLULAR MICROBIOLOGY
- The Beclin 1 interactome
- (2010) Congcong He et al. CURRENT OPINION IN CELL BIOLOGY
- Autophagy requires endoplasmic reticulum targeting of the PI3-kinase complex via Atg14L
- (2010) Kohichi Matsunaga et al. JOURNAL OF CELL BIOLOGY
- Autophagy and innate immunity: Triggering, targeting and tuning
- (2010) Rhea Sumpter et al. SEMINARS IN CELL & DEVELOPMENTAL BIOLOGY
- Anaplasma phagocytophilum Ats-1 Is Imported into Host Cell Mitochondria and Interferes with Apoptosis Induction
- (2010) Hua Niu et al. PLoS Pathogens
- Biological Diversity of Prokaryotic Type IV Secretion Systems
- (2009) C. E. Alvarez-Martinez et al. MICROBIOLOGY AND MOLECULAR BIOLOGY REVIEWS
- Distinct regulation of autophagic activity by Atg14L and Rubicon associated with Beclin 1–phosphatidylinositol-3-kinase complex
- (2009) Yun Zhong et al. NATURE CELL BIOLOGY
- Two Beclin 1-binding proteins, Atg14L and Rubicon, reciprocally regulate autophagy at different stages
- (2009) Kohichi Matsunaga et al. NATURE CELL BIOLOGY
- Human Granulocytic Anaplasmosis
- (2008) Johan S. Bakken et al. INFECTIOUS DISEASE CLINICS OF NORTH AMERICA
- Autophagosome formation from membrane compartments enriched in phosphatidylinositol 3-phosphate and dynamically connected to the endoplasmic reticulum
- (2008) Elizabeth L. Axe et al. JOURNAL OF CELL BIOLOGY
- The mitochondrial targeting sequence tilts the balance between mitochondrial and cytosolic dual localization
- (2008) N. Regev-Rudzki et al. JOURNAL OF CELL SCIENCE
- Subversion of cellular autophagy by Anaplasma phagocytophilum
- (2007) Hua Niu et al. CELLULAR MICROBIOLOGY
Find the ideal target journal for your manuscript
Explore over 38,000 international journals covering a vast array of academic fields.
SearchAsk a Question. Answer a Question.
Quickly pose questions to the entire community. Debate answers and get clarity on the most important issues facing researchers.
Get Started