Isolated C-terminal tail of FGF23 alleviates hypophosphatemia by inhibiting FGF23-FGFR-Klotho complex formation

Inactivation of klotho function induces hyperphosphatemia even in presence of high serum fibroblast growth factor 23 levels in a genetically engineered hypophosphatemic (Hyp) mouse model

(2009) Teruyo Nakatani et al.   FASEB JOURNAL

Therapeutic Effects of Anti-FGF23 Antibodies in Hypophosphatemic Rickets/Osteomalacia*

(2009) Yukiko Aono et al.   JOURNAL OF BONE AND MINERAL RESEARCH

FGF23 Elevation and Hypophosphatemia after Intravenous Iron Polymaltose: A Prospective Study

(2009) Belinda J. Schouten et al.   JOURNAL OF CLINICAL ENDOCRINOLOGY & METABOLISM

Initial FGF23-Mediated Signaling Occurs in the Distal Convoluted Tubule

(2009) E. G. Farrow et al.   JOURNAL OF THE AMERICAN SOCIETY OF NEPHROLOGY

Molecular genetic and biochemical analyses of FGF23 mutations in familial tumoral calcinosis

(2008) Holly J. Garringer et al.   AMERICAN JOURNAL OF PHYSIOLOGY-ENDOCRINOLOGY AND METABOLISM

Calcimimetics as an Adjuvant Treatment for Familial Hypophosphatemic Rickets

(2008) U. S. Alon et al.   Clinical Journal of the American Society of Nephrology

Tumor producing fibroblast growth factor 23 localized by two-staged venous sampling

(2008) Gerben van Boekel et al.   EUROPEAN JOURNAL OF ENDOCRINOLOGY

Anti-FGF23 Neutralizing Antibodies Show the Physiological Role and Structural Features of FGF23

(2008) Yuji Yamazaki et al.   JOURNAL OF BONE AND MINERAL RESEARCH