Hypertension prevents a sensory stimulation-based collateral therapeutic from protecting the cortex from impending ischemic stroke damage in a spontaneously hypersensitive rat model

Stent-Retriever Thrombectomy after Intravenous t-PA vs. t-PA Alone in Stroke

(2015) Jeffrey L. Saver et al.   NEW ENGLAND JOURNAL OF MEDICINE

Hypertension

(2013) Giuseppe Faraco et al.   HYPERTENSION

Multimodal Imaging in Rats Reveals Impaired Neurovascular Coupling in Sustained Hypertension

(2013) Novella Calcinaghi et al.   STROKE

Central Cardiovascular Circuits Contribute to the Neurovascular Dysfunction in Angiotensin II Hypertension

(2012) C. Capone et al.   JOURNAL OF NEUROSCIENCE

Mild Sensory Stimulation Reestablishes Cortical Function during the Acute Phase of Ischemia

(2011) C. C. Lay et al.   JOURNAL OF NEUROSCIENCE

Amount but Not Pattern of Protective Sensory Stimulation Alters Recovery After Permanent Middle Cerebral Artery Occlusion

(2011) Melissa F. Davis et al.   STROKE

The cerebrovascular dysfunction induced by slow pressor doses of angiotensin II precedes the development of hypertension

(2010) Carmen Capone et al.   AMERICAN JOURNAL OF PHYSIOLOGY-HEART AND CIRCULATORY PHYSIOLOGY

Mild Sensory Stimulation Completely Protects the Adult Rodent Cortex from Ischemic Stroke

(2010) Christopher C. Lay et al.   PLoS One

The Pattern of Leptomeningeal Collaterals on CT Angiography Is a Strong Predictor of Long-Term Functional Outcome in Stroke Patients With Large Vessel Intracranial Occlusion

(2010) Fabricio O. Lima et al.   STROKE

Update of the Stroke Therapy Academic Industry Roundtable Preclinical Recommendations

(2009) Marc Fisher et al.   STROKE

Hypertension and Cerebrovascular Dysfunction

(2008) Costantino Iadecola et al.   Cell Metabolism