Restored nitric oxide bioavailability reduces the severity of acute-to-chronic transition in a mouse model of aristolochic acid nephropathy
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Title
Restored nitric oxide bioavailability reduces the severity of acute-to-chronic transition in a mouse model of aristolochic acid nephropathy
Authors
Keywords
Kidneys, Nitric oxide, Mouse models, Fibrosis, Chronic kidney disease, Renal system, Inflammation, Macrophages
Journal
PLoS One
Volume 12, Issue 8, Pages e0183604
Publisher
Public Library of Science (PLoS)
Online
2017-08-24
DOI
10.1371/journal.pone.0183604
References
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Note: Only part of the references are listed.- An Integrated View of Aristolochic Acid Nephropathy: Update of the Literature
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- Acute Kidney Injury and Chronic Kidney Disease as Interconnected Syndromes
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- Aristolochic acid-induced accumulation of methylglyoxal and Nε-(carboxymethyl)lysine: An important and novel pathway in the pathogenic mechanism for aristolochic acid nephropathy
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- Aristolochic acid nephropathy revisited: a place for innate and adaptive immunity?
- (2010) Agnieszka A Pozdzik et al. HISTOPATHOLOGY
- Asymmetric dimethylarginine (ADMA) induces chronic kidney disease through a mechanism involving collagen and TGF-β1 synthesis
- (2010) Fabrice Mihout et al. JOURNAL OF PATHOLOGY
- Aristolochic acid nephropathy: A worldwide problem
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- Patterns of interstitial inflammation during the evolution of renal injury in experimental aristolochic acid nephropathy
- (2008) A. A. Pozdzik et al. NEPHROLOGY DIALYSIS TRANSPLANTATION
- Ischemic injury underlies the pathogenesis of aristolochic acid−induced acute kidney injury
- (2008) Yu-Jie Wen et al. Translational Research
- Aristolochic acid induces proximal tubule apoptosis and epithelial to mesenchymal transformation
- (2007) A.A. Pozdzik et al. KIDNEY INTERNATIONAL
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