Interleukin-17A-induced production of acute serum amyloid A by keratinocytes contributes to psoriasis pathogenesis
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Title
Interleukin-17A-induced production of acute serum amyloid A by keratinocytes contributes to psoriasis pathogenesis
Authors
Keywords
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Journal
PLoS One
Volume 12, Issue 7, Pages e0181486
Publisher
Public Library of Science (PLoS)
Online
2017-07-15
DOI
10.1371/journal.pone.0181486
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Related references
Note: Only part of the references are listed.- The Involvement of Serum Amyloid A in Psoriatic Inflammation
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- Serum amyloid A induces interleukin-1β secretion from keratinocytes via the NACHT, LRR and PYD domains-containing protein 3 inflammasome
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- IMQ-induced skin inflammation in mice is dependent on IL-1R1 and MyD88 signaling but independent of the NLRP3 inflammasome
- (2015) Hanitriniaina Rabeony et al. EUROPEAN JOURNAL OF IMMUNOLOGY
- TLR2 and TLR4 expression in atopic dermatitis, contact dermatitis and psoriasis
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- Cutting Edge: TLR2 Is a Functional Receptor for Acute-Phase Serum Amyloid A
- (2014) N. Cheng et al. JOURNAL OF IMMUNOLOGY
- Inhibition of Keratinocyte Differentiation by the Synergistic Effect of IL-17A, IL-22, IL-1α, TNFα and Oncostatin M
- (2014) Hanitriniaina Rabeony et al. PLoS One
- Role of formyl peptide receptor 2 on the serum amyloid A-induced macrophage foam cell formation
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- Smoking and the skin
- (2012) Arisa Ortiz et al. INTERNATIONAL JOURNAL OF DERMATOLOGY
- Smoking and pathogenesis of psoriasis: a review of oxidative, inflammatory and genetic mechanisms
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- Is serum amyloid A protein a better indicator of inflammation in severe psoriasis?
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- CD36 Is a Novel Serum Amyloid A (SAA) Receptor Mediating SAA Binding and SAA-induced Signaling in Human and Rodent Cells
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- CCL20 and -Defensin-2 Induce Arrest of Human Th17 Cells on Inflamed Endothelium In Vitro under Flow Conditions
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- Imiquimod-Induced Psoriasis-Like Skin Inflammation in Mice Is Mediated via the IL-23/IL-17 Axis
- (2009) L. van der Fits et al. JOURNAL OF IMMUNOLOGY
- AGE-Modified Collagens I and III Induce Keratinocyte Terminal Differentiation through AGE Receptor CD36: Epidermal–Dermal Interaction in Acquired Perforating Dermatosis
- (2009) Eita Fujimoto et al. JOURNAL OF INVESTIGATIVE DERMATOLOGY
- Psoriasis
- (2009) Frank O. Nestle et al. NEW ENGLAND JOURNAL OF MEDICINE
- Is serum amyloid A an endogenous TLR4 agonist?
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