C-Type Lectin Receptor Dectin-2 Binds to an Endogenous Protein β-Glucuronidase on Dendritic Cells
Published 2017 View Full Article
- Home
- Publications
- Publication Search
- Publication Details
Title
C-Type Lectin Receptor Dectin-2 Binds to an Endogenous Protein β-Glucuronidase on Dendritic Cells
Authors
Keywords
Mannose, Glycosylation, Fungal structure, Lectins, Pattern recognition receptors, Immune receptors, Macrophages, Protein structure
Journal
PLoS One
Volume 12, Issue 1, Pages e0169562
Publisher
Public Library of Science (PLoS)
Online
2017-01-10
DOI
10.1371/journal.pone.0169562
References
Ask authors/readers for more resources
Related references
Note: Only part of the references are listed.- Dectin-2 Recognizes Mannosylated O-antigens of Human Opportunistic Pathogens and Augments Lipopolysaccharide Activation of Myeloid Cells
- (2016) Alexandra Wittmann et al. JOURNAL OF BIOLOGICAL CHEMISTRY
- C-type lectins in immunity: recent developments
- (2015) Ivy M Dambuza et al. CURRENT OPINION IN IMMUNOLOGY
- Recent insights into structures and functions of C-type lectins in the immune system
- (2015) Kurt Drickamer et al. CURRENT OPINION IN STRUCTURAL BIOLOGY
- Human Mincle Binds to Cholesterol Crystals and Triggers Innate Immune Responses
- (2015) Ryoko Kiyotake et al. JOURNAL OF BIOLOGICAL CHEMISTRY
- Dectin-2 Is a Direct Receptor for Mannose-Capped Lipoarabinomannan of Mycobacteria
- (2014) Akiko Yonekawa et al. IMMUNITY
- Bone marrow-derived macrophages from BALB/c and C57BL/6 mice fundamentally differ in their respiratory chain complex proteins, lysosomal enzymes and components of antioxidant stress systems
- (2014) Maren Depke et al. Journal of Proteomics
- Identification of Distinct Ligands for the C-type Lectin Receptors Mincle and Dectin-2 in the Pathogenic Fungus Malassezia
- (2013) Tetsuaki Ishikawa et al. Cell Host & Microbe
- F-Actin Is an Evolutionarily Conserved Damage-Associated Molecular Pattern Recognized by DNGR-1, a Receptor for Dead Cells
- (2012) Susan Ahrens et al. IMMUNITY
- The Dendritic Cell Receptor Clec9A Binds Damaged Cells via Exposed Actin Filaments
- (2012) Jian-Guo Zhang et al. IMMUNITY
- Vertebrate protein glycosylation: diversity, synthesis and function
- (2012) Kelley W. Moremen et al. NATURE REVIEWS MOLECULAR CELL BIOLOGY
- Control of TNF-Induced Dendritic Cell Maturation by Hybrid-Type N-Glycans
- (2011) S. Schlickeiser et al. JOURNAL OF IMMUNOLOGY
- Overview of the mucopolysaccharidoses
- (2011) J. Muenzer RHEUMATOLOGY
- Dectin-2 Recognition of α-Mannans and Induction of Th17 Cell Differentiation Is Essential for Host Defense against Candida albicans
- (2010) Shinobu Saijo et al. IMMUNITY
- Cutting Edge: Mincle Is Essential for Recognition and Adjuvanticity of the Mycobacterial Cord Factor and its Synthetic Analog Trehalose-Dibehenate
- (2010) H. Schoenen et al. JOURNAL OF IMMUNOLOGY
- Dectin-2 is a Syk-coupled pattern recognition receptor crucial for Th17 responses to fungal infection
- (2009) Matthew J. Robinson et al. JOURNAL OF EXPERIMENTAL MEDICINE
- Direct recognition of the mycobacterial glycolipid, trehalose dimycolate, by C-type lectin Mincle
- (2009) Eri Ishikawa et al. JOURNAL OF EXPERIMENTAL MEDICINE
- Involvement of the Toll-like receptor 4 pathway and use of TNF- antagonists for treatment of the mucopolysaccharidoses
- (2009) C. M. Simonaro et al. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA
- Pattern recognition receptors and control of adaptive immunity
- (2008) Noah W. Palm et al. IMMUNOLOGICAL REVIEWS
- Mincle is an ITAM-coupled activating receptor that senses damaged cells
- (2008) Sho Yamasaki et al. NATURE IMMUNOLOGY
- Early Neurodegeneration Progresses Independently of Microglial Activation by Heparan Sulfate in the Brain of Mucopolysaccharidosis IIIB Mice
- (2008) Jérôme Ausseil et al. PLoS One
- Mechanism of Glycosaminoglycan-Mediated Bone and Joint Disease
- (2007) Calogera M. Simonaro et al. AMERICAN JOURNAL OF PATHOLOGY
Publish scientific posters with Peeref
Peeref publishes scientific posters from all research disciplines. Our Diamond Open Access policy means free access to content and no publication fees for authors.
Learn MoreAsk a Question. Answer a Question.
Quickly pose questions to the entire community. Debate answers and get clarity on the most important issues facing researchers.
Get Started