Inhibition of the Unfolded Protein Response Mechanism Prevents Cardiac Fibrosis

HCV induces transforming growth factor β1 through activation of endoplasmic reticulum stress and the unfolded protein response

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Diabetes Mellitus in Outpatients in Debre Berhan Referral Hospital, Ethiopia

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Islands of spatially discordant APD alternans underlie arrhythmogenesis by promoting electrotonic dyssynchrony in models of fibrotic rat ventricular myocardium

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Keep your heart in shape: molecular chaperone networks for treating heart disease

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The Inhibitory Effect of Quercetin on Asymmetric Dimethylarginine-Induced Apoptosis Is Mediated by the Endoplasmic Reticulum Stress Pathway in Glomerular Endothelial Cells

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Epithelial Calreticulin Up-Regulation Promotes Profibrotic Responses and Tubulointerstitial Fibrosis Development

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Calreticulin Induces Dilated Cardiomyopathy

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The role of endoplasmic reticulum stress and the unfolded protein response in fibrosis

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New concepts of endoplasmic reticulum function in the heart: Programmed to conserve

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Deletion of periostin reduces muscular dystrophy and fibrosis in mice by modulating the transforming growth factor-  pathway

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Cardiac Sarcoplasmic Reticulum Calcium Leak: Basis and Roles in Cardiac Dysfunction

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Assessment of Myocardial Fibrosis With Cardiovascular Magnetic Resonance

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The Unfolded Protein Response: From Stress Pathway to Homeostatic Regulation

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Potential Therapeutic Targets for Cardiac Fibrosis

(2010) Andrew Leask   CIRCULATION RESEARCH

Intracellular Calreticulin Regulates Multiple Steps in Fibrillar Collagen Expression, Trafficking, and Processing into the Extracellular Matrix

(2010) Lauren Van Duyn Graham et al.   JOURNAL OF BIOLOGICAL CHEMISTRY

Calreticulin, a multi-process calcium-buffering chaperone of the endoplasmic reticulum

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From endoplasmic-reticulum stress to the inflammatory response

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Altered expression of calreticulin during the development of fibrosis

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