Hyperglycemia Induces Cellular Hypoxia through Production of Mitochondrial ROS Followed by Suppression of Aquaporin-1

NA+/H+ Exchanger 1-and Aquaporin-1-Dependent Hyperosmolarity Changes Decrease Nitric Oxide Production and Induce VCAM-1 Expression in Endothelial Cells Exposed to High Glucose

(2017) R. Madonna et al.   INTERNATIONAL JOURNAL OF IMMUNOPATHOLOGY AND PHARMACOLOGY

Aquaporin-1 and Sodium-Hydrogen Exchangers as Pharmacological Targets in Diabetic Atherosclerosis

(2015) Rosalinda Madonna et al.   CURRENT DRUG TARGETS

Cellular Hypoxia of Pancreatic β-Cells Due to High Levels of Oxygen Consumption for Insulin Secretionin Vitro

(2011) Yoshifumi Sato et al.   JOURNAL OF BIOLOGICAL CHEMISTRY

Phosphorescent Light–Emitting Iridium Complexes Serve as a Hypoxia-Sensing Probe for Tumor Imaging in Living Animals

(2010) Shaojuan Zhang et al.   CANCER RESEARCH

Activation of Protein Kinase C Isoforms and Its Impact on Diabetic Complications

(2010) Pedro Geraldes et al.   CIRCULATION RESEARCH

Endothelial MnSOD overexpression prevents retinal VEGF expression in diabetic mice

(2007) Hideo Goto et al.   BIOCHEMICAL AND BIOPHYSICAL RESEARCH COMMUNICATIONS