Analyzing and Modeling the Kinetics of Amyloid Beta Pores Associated with Alzheimer’s Disease Pathology
Published 2015 View Full Article
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Title
Analyzing and Modeling the Kinetics of Amyloid Beta Pores Associated with Alzheimer’s Disease Pathology
Authors
Keywords
Permeability, Oocytes, Markov models, Fluorescence imaging, Membrane potential, Oligomers, Alzheimers disease, Cell membranes
Journal
PLoS One
Volume 10, Issue 9, Pages e0137357
Publisher
Public Library of Science (PLoS)
Online
2015-09-09
DOI
10.1371/journal.pone.0137357
References
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- (2014) Alan L. Gillman et al. JOURNAL OF PHYSICAL CHEMISTRY B
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- (2012) Ghanim Ullah et al. JOURNAL OF GENERAL PHYSIOLOGY
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- Constitutive cAMP response element binding protein (CREB) activation by Alzheimer's disease presenilin-driven inositol trisphosphate receptor (InsP3R) Ca2+signaling
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- (2010) Angelo Demuro et al. JOURNAL OF BIOLOGICAL CHEMISTRY
- Calcium Signalling and Alzheimer’s Disease
- (2010) Michael J. Berridge NEUROCHEMICAL RESEARCH
- Deleterious Effects of Amyloid β Oligomers Acting as an Extracellular Scaffold for mGluR5
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- (2009) Michael J. Berridge PFLUGERS ARCHIV-EUROPEAN JOURNAL OF PHYSIOLOGY
- Small molecule blockers of the Alzheimer A calcium channel potently protect neurons from A cytotoxicity
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- (2009) Ilya Bezprozvanny TRENDS IN MOLECULAR MEDICINE
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- (2008) Kim N. Green et al. NEURON
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- (2008) Ilya Bezprozvanny et al. TRENDS IN NEUROSCIENCES
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