Molecular Mechanisms of 2, 3′, 4, 4′, 5-Pentachlorobiphenyl-Induced Thyroid Dysfunction in FRTL-5 Cells
Published 2015 View Full Article
- Home
- Publications
- Publication Search
- Publication Details
Title
Molecular Mechanisms of 2, 3′, 4, 4′, 5-Pentachlorobiphenyl-Induced Thyroid Dysfunction in FRTL-5 Cells
Authors
Keywords
Apoptosis, Small interfering RNAs, Thyroid, Forkhead box, Gene expression, Luciferase, Transfection, Mixtures
Journal
PLoS One
Volume 10, Issue 3, Pages e0120133
Publisher
Public Library of Science (PLoS)
Online
2015-03-20
DOI
10.1371/journal.pone.0120133
References
Ask authors/readers for more resources
Related references
Note: Only part of the references are listed.- Dual PI3K/mTOR inhibition is required to effectively impair microenvironment survival signals in mantle cell lymphoma
- (2015) Laia Rosich et al. Oncotarget
- The Acute Inhibitory Effect of Iodide Excess on Sodium/Iodide Symporter Expression and Activity Involves the PI3K/Akt Signaling Pathway
- (2014) Caroline Serrano-Nascimento et al. ENDOCRINOLOGY
- Wnt-Independent Role of β-Catenin in Thyroid Cell Proliferation and Differentiation
- (2014) Ana Sastre-Perona et al. MOLECULAR ENDOCRINOLOGY
- Morphological and functional deterioration of the rat thyroid following chronic exposure to low-dose PCB118
- (2013) Jin-Mei Tang et al. EXPERIMENTAL AND TOXICOLOGIC PATHOLOGY
- Development of PI3K inhibitors: lessons learned from early clinical trials
- (2013) Jordi Rodon et al. Nature Reviews Clinical Oncology
- The sodium/iodide symporter NIS is a transcriptional target of the p53-family members in liver cancer cells
- (2013) F Guerrieri et al. Cell Death & Disease
- Modulation of sodium iodide symporter expression and function by LY294002, Akti-1/2 and Rapamycin in thyroid cells
- (2012) Yu-Yu Liu et al. ENDOCRINE-RELATED CANCER
- Induction of Sodium/Iodide Symporter (NIS) Expression and Radioiodine Uptake in Non-Thyroid Cancer Cells
- (2012) Zhi Liu et al. PLoS One
- Down-Regulation of AP-4 Inhibits Proliferation, Induces Cell Cycle Arrest and Promotes Apoptosis in Human Gastric Cancer Cells
- (2012) Xinghua Liu et al. PLoS One
- Fragments of Genomic DNA Released by Injured Cells Activate Innate Immunity and Suppress Endocrine Function in the Thyroid
- (2011) Akira Kawashima et al. ENDOCRINOLOGY
- FOXO3 Is Inhibited by Oncogenic PI3K/Akt Signaling but Can Be Reactivated by the NSAID Sulindac Sulfide
- (2011) Carl Weidinger et al. JOURNAL OF CLINICAL ENDOCRINOLOGY & METABOLISM
- Non-dioxin-like PCBs interact with benzo[a]pyrene-induced p53-responses and inhibit apoptosis
- (2010) Lauy Al-Anati et al. TOXICOLOGY AND APPLIED PHARMACOLOGY
- PCB-153 exposure coordinates cell cycle progression and cellular metabolism in human mammary epithelial cells
- (2010) Venkatasubbaiah A. Venkatesha et al. TOXICOLOGY LETTERS
- Weight of Evidence Evaluation of Potential Human Cancer Risks from Exposure to Polychlorinated Biphenyls: An Update Based on Studies Published Since 2003
- (2009) Robert Golden et al. CRITICAL REVIEWS IN TOXICOLOGY
- Mutual Dependence of Foxo3a and PGC-1α in the Induction of Oxidative Stress Genes
- (2009) Yolanda Olmos et al. JOURNAL OF BIOLOGICAL CHEMISTRY
- Burdens of PBBs, PBDEs, and PCBs in tissues of the cancer patients in the e-waste disassembly sites in Zhejiang, China
- (2009) Gaofeng Zhao et al. SCIENCE OF THE TOTAL ENVIRONMENT
- Distinct functional significance of Akt and mTOR constitutive activation in mantle cell lymphoma
- (2008) J. Dal Col et al. BLOOD
- Phosphoinositide-3-kinase inhibition induces sodium/iodide symporter expression in rat thyroid cells and human papillary thyroid cancer cells
- (2008) Takahiko Kogai et al. JOURNAL OF ENDOCRINOLOGY
- Gβγ Dimers Released in Response to Thyrotropin Activate Phosphoinositide 3-Kinase and Regulate Gene Expression in Thyroid Cells
- (2008) Miguel A. Zaballos et al. MOLECULAR ENDOCRINOLOGY
Discover Peeref hubs
Discuss science. Find collaborators. Network.
Join a conversationBecome a Peeref-certified reviewer
The Peeref Institute provides free reviewer training that teaches the core competencies of the academic peer review process.
Get Started