Alzheimer's Associated β-Amyloid Protein Inhibits Influenza A Virus and Modulates Viral Interactions with Phagocytes
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Title
Alzheimer's Associated β-Amyloid Protein Inhibits Influenza A Virus and Modulates Viral Interactions with Phagocytes
Authors
Keywords
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Journal
PLoS One
Volume 9, Issue 7, Pages e101364
Publisher
Public Library of Science (PLoS)
Online
2014-07-03
DOI
10.1371/journal.pone.0101364
References
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- (2012) Alexander Slowik et al. Molecular Neurodegeneration
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- (2011) Jingdong Zhang et al. Journal of Neuroimmune Pharmacology
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- The ability of pandemic influenza virus hemagglutinins to induce lower respiratory pathology is associated with decreased surfactant protein D binding
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- The Alzheimer's Disease-Associated Amyloid β-Protein Is an Antimicrobial Peptide
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- APP Processing Induced by Herpes Simplex Virus Type 1 (HSV-1) Yields Several APP Fragments in Human and Rat Neuronal Cells
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- Interactions of -, -, and -Defensins with Influenza A Virus and Surfactant Protein D
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- Models of Toxic β-Sheet Channels of Protegrin-1 Suggest a Common Subunit Organization Motif Shared with Toxic Alzheimer β-Amyloid Ion Channels
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- Toll-Like Receptor 2 Acts as a Natural Innate Immune Receptor to Clear Amyloid 1-42 and Delay the Cognitive Decline in a Mouse Model of Alzheimer's Disease
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