Contribution of a Non-β-Cell Source to β-Cell Mass during Pregnancy
Published 2014 View Full Article
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Title
Contribution of a Non-β-Cell Source to β-Cell Mass during Pregnancy
Authors
Keywords
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Journal
PLoS One
Volume 9, Issue 6, Pages e100398
Publisher
Public Library of Science (PLoS)
Online
2014-06-19
DOI
10.1371/journal.pone.0100398
References
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Note: Only part of the references are listed.- No evidence for β cell neogenesis in murine adult pancreas
- (2013) Xiangwei Xiao et al. JOURNAL OF CLINICAL INVESTIGATION
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- (2013) M Van de Casteele et al. Cell Death & Disease
- Pancreatic β Cell Dedifferentiation as a Mechanism of Diabetic β Cell Failure
- (2012) Chutima Talchai et al. CELL
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- (2012) D. E. Metzger et al. DEVELOPMENT
- Plasticity of Adult Human Pancreatic Duct Cells by Neurogenin3-Mediated Reprogramming
- (2012) Nathalie Swales et al. PLoS One
- Sox9-Haploinsufficiency Causes Glucose Intolerance in Mice
- (2011) Claire L. Dubois et al. PLoS One
- Non-β-cell progenitors of β-cells in pregnant mice
- (2011) Sylvie Abouna et al. Organogenesis
- Pancreatic β Cells Require NeuroD to Achieve and Maintain Functional Maturity
- (2010) Chunyan Gu et al. Cell Metabolism
- -Cell Growth and Regeneration: Replication Is Only Part of the Story
- (2010) S. Bonner-Weir et al. DIABETES
- Adaptive changes in pancreatic beta cell fractional area and beta cell turnover in human pregnancy
- (2010) A. E. Butler et al. DIABETOLOGIA
- Conversion of adult pancreatic α-cells to β-cells after extreme β-cell loss
- (2010) Fabrizio Thorel et al. NATURE
- Rfx6 directs islet formation and insulin production in mice and humans
- (2010) Stuart B. Smith et al. NATURE
- Neurogenin3 inhibits proliferation in endocrine progenitors by inducing Cdkn1a
- (2010) T. Miyatsuka et al. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA
- The Ectopic Expression of Pax4 in the Mouse Pancreas Converts Progenitor Cells into α and Subsequently β Cells
- (2009) Patrick Collombat et al. CELL
- Neurog3 gene dosage regulates allocation of endocrine and exocrine cell fates in the developing mouse pancreas
- (2009) Sui Wang et al. DEVELOPMENTAL BIOLOGY
- Adult human β-cell neogenesis?
- (2009) C. Demeterco et al. DIABETES OBESITY & METABOLISM
- Prolactin Receptors Are Critical to the Adaptation of Islets to Pregnancy
- (2009) Robert L. Sorenson et al. ENDOCRINOLOGY
- Sustained Neurog3 expression in hormone-expressing islet cells is required for endocrine maturation and function
- (2009) S. Wang et al. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA
- Transdifferentiation of pancreatic ductal cells to endocrine β-cells
- (2008) Susan Bonner-Weir et al. BIOCHEMICAL SOCIETY TRANSACTIONS
- β Cells Can Be Generated from Endogenous Progenitors in Injured Adult Mouse Pancreas
- (2008) Xiaobo Xu et al. CELL
- HES-1 Is Involved in Adaptation of Adult Human -Cells to Proliferation In Vitro
- (2008) Y. Bar et al. DIABETES
- Maturation of Adult β-Cells Revealed Using aPdx1/InsulinDual-Reporter Lentivirus
- (2008) Marta Szabat et al. ENDOCRINOLOGY
- Prolactin Receptor Is Required for Normal Glucose Homeostasis and Modulation of β-Cell Mass during Pregnancy
- (2008) Carol Huang et al. ENDOCRINOLOGY
- On the origin of the cell
- (2008) J. M. Oliver-Krasinski et al. GENES & DEVELOPMENT
- Carbonic anhydrase II-positive pancreatic cells are progenitors for both endocrine and exocrine pancreas after birth
- (2008) A. Inada et al. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA
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