DLK1 Promotes Lung Cancer Cell Invasion through Upregulation of MMP9 Expression Depending on Notch Signaling
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Title
DLK1 Promotes Lung Cancer Cell Invasion through Upregulation of MMP9 Expression Depending on Notch Signaling
Authors
Keywords
Notch signaling, Lung and intrathoracic tumors, Gene expression, Hyperexpression techniques, Metastasis, Small interfering RNAs, Extracellular matrix, Non-small cell lung cancer
Journal
PLoS One
Volume 9, Issue 3, Pages e91509
Publisher
Public Library of Science (PLoS)
Online
2014-03-13
DOI
10.1371/journal.pone.0091509
References
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Related references
Note: Only part of the references are listed.- Possible roles of DLK1 in the Notch pathway during development and disease
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- DLK1 as a Potential Target against Cancer Stem/Progenitor Cells of Hepatocellular Carcinoma
- (2012) X. Xu et al. MOLECULAR CANCER THERAPEUTICS
- The EGF-like proteins DLK1 and DLK2 function as inhibitory non-canonical ligands of NOTCH1 receptor that modulate each other's activities
- (2011) Beatriz Sánchez-Solana et al. BIOCHIMICA ET BIOPHYSICA ACTA-MOLECULAR CELL RESEARCH
- DLK1, delta-like 1 homolog (Drosophila), regulates tumor cell differentiation in vivo
- (2011) Asma Begum et al. CANCER LETTERS
- Prognostic impact of Notch ligands and receptors in nonsmall cell lung cancer
- (2010) Tom Donnem et al. CANCER
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- Notch-1 stimulates survival of lung adenocarcinoma cells during hypoxia by activating the IGF-1R pathway
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- Pref-1 Regulates Mesenchymal Cell Commitment and Differentiation through Sox9
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- Targeted Knockdown of Notch1 Inhibits Invasion of Human Prostate Cancer Cells Concomitant with Inhibition of Matrix Metalloproteinase-9 and Urokinase Plasminogen Activator
- (2009) B. Bin Hafeez et al. CLINICAL CANCER RESEARCH
- Minireview: Pref-1: Role in Adipogenesis and Mesenchymal Cell Fate
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- The atypical mammalian ligand Delta-like1 (Dlk1) can regulate Notch signalling in Drosophila
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- Delta-Like 1 (Dlk-1), a Novel Marker of Prostate Basal and Candidate Epithelial Stem Cells, Is Downregulated by Notch Signalling in Intermediate/Transit Amplifying Cells of the Human Prostate
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- dlk1 Specifically Interacts with Insulin-Like Growth Factor Binding Protein 1 to Modulate Adipogenesis of 3T3-L1 Cells
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