ER-α36-Mediated Rapid Estrogen Signaling Positively Regulates ER-Positive Breast Cancer Stem/Progenitor Cells
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Title
ER-α36-Mediated Rapid Estrogen Signaling Positively Regulates ER-Positive Breast Cancer Stem/Progenitor Cells
Authors
Keywords
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Journal
PLoS One
Volume 9, Issue 2, Pages e88034
Publisher
Public Library of Science (PLoS)
Online
2014-02-19
DOI
10.1371/journal.pone.0088034
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Note: Only part of the references are listed.- Estrogen Receptor-α Variant, ER-α36, is Involved in Tamoxifen Resistance and Estrogen Hypersensitivity
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- ERα36, a new variant of the ERα is expressed in triple negative breast carcinomas and has a specific transcriptomic signature in breast cancer cell lines
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- The Tumor Suppressor p53 Regulates Polarity of Self-Renewing Divisions in Mammary Stem Cells
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- Expression of ER-α36, a Novel Variant of Estrogen Receptor α, and Resistance to Tamoxifen Treatment in Breast Cancer
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- Aberrant luminal progenitors as the candidate target population for basal tumor development in BRCA1 mutation carriers
- (2009) Elgene Lim et al. NATURE MEDICINE
- Regulation of Mammary Stem/Progenitor Cells by PTEN/Akt/β-Catenin Signaling
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- (2008) H Korkaya et al. ONCOGENE
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