Acetaldehyde-Derived Advanced Glycation End-Products Promote Alcoholic Liver Disease

Involvement of the Toxic AGEs (TAGE)-RAGE System in the Pathogenesis of Diabetic Vascular Complications: A Novel Therapeutic Strategy

(2012) Masayoshi Takeuchi et al.   CURRENT DRUG TARGETS

Advanced glycation end products and neurodegenerative diseases: Mechanisms and perspective

(2012) Jinlong Li et al.   JOURNAL OF THE NEUROLOGICAL SCIENCES

Alcoholic Liver Disease: Pathogenesis and New Therapeutic Targets

(2011) Bin Gao et al.   GASTROENTEROLOGY

Mechanisms and cell signaling in alcoholic liver disease

(2010) Juliane I. Beier et al.   BIOLOGICAL CHEMISTRY

Atorvastatin decreases serum levels of advanced glycation endproducts (AGEs) in nonalcoholic steatohepatitis (NASH) patients with dyslipidemia: clinical usefulness of AGEs as a biomarker for the attenuation of NASH

(2010) Yuki Kimura et al.   JOURNAL OF GASTROENTEROLOGY

Acetaldehyde Adducts in Alcoholic Liver Disease

(2010) Mashiko Setshedi et al.   Oxidative Medicine and Cellular Longevity

Role of oxidative stress in alcohol-induced liver injury

(2009) Arthur I. Cederbaum et al.   ARCHIVES OF TOXICOLOGY

Oxidative Stress and Alcoholic Liver Disease

(2009) Defeng Wu et al.   SEMINARS IN LIVER DISEASE

CYP2E1 and oxidative liver injury by alcohol

(2007) Yongke Lu et al.   FREE RADICAL BIOLOGY AND MEDICINE