Impaired Macrophage and Satellite Cell Infiltration Occurs in a Muscle-Specific Fashion Following Injury in Diabetic Skeletal Muscle
Published 2013 View Full Article
- Home
- Publications
- Publication Search
- Publication Details
Title
Impaired Macrophage and Satellite Cell Infiltration Occurs in a Muscle-Specific Fashion Following Injury in Diabetic Skeletal Muscle
Authors
Keywords
Muscle regeneration, Skeletal muscles, Diabetes mellitus, Soleus muscles, Collagens, Gastrocnemius muscles, Musculoskeletal injury, Macrophages
Journal
PLoS One
Volume 8, Issue 8, Pages e70971
Publisher
Public Library of Science (PLoS)
Online
2013-08-13
DOI
10.1371/journal.pone.0070971
References
Ask authors/readers for more resources
Related references
Note: Only part of the references are listed.- Urokinase-Type Plasminogen Activator Plays Essential Roles in Macrophage Chemotaxis and Skeletal Muscle Regeneration
- (2014) S. C. Bryer et al. JOURNAL OF IMMUNOLOGY
- Matrix Metalloproteinase-9 Deficiency Results in Decreased Fiber Cross-Sectional Area and Alters Fiber Type Distribution in Mouse Hindlimb Skeletal Muscle
- (2011) Ryan S. Mehan et al. CELLS TISSUES ORGANS
- Inhibition of Plasminogen Activator Inhibitor-1 Restores Skeletal Muscle Regeneration in Untreated Type 1 Diabetic Mice
- (2011) M. P. Krause et al. DIABETES
- Macrophage-Specific Expression of Urokinase-Type Plasminogen Activator Promotes Skeletal Muscle Regeneration
- (2011) M. L. Novak et al. JOURNAL OF IMMUNOLOGY
- Regulatory interactions between muscle and the immune system during muscle regeneration
- (2010) James G. Tidball et al. AMERICAN JOURNAL OF PHYSIOLOGY-REGULATORY INTEGRATIVE AND COMPARATIVE PHYSIOLOGY
- PTEN Inhibition Improves Muscle Regeneration in Mice Fed a High-Fat Diet
- (2010) Z. Hu et al. DIABETES
- Effects of type 1 diabetes mellitus on skeletal muscle: clinical observations and physiological mechanisms
- (2010) Matthew P Krause et al. PEDIATRIC DIABETES
- Impaired Growth and Force Production in Skeletal Muscles of Young Partially Pancreatectomized Rats: A Model of Adolescent Type 1 Diabetic Myopathy?
- (2010) Carly S. Gordon et al. PLoS One
- Plasminogen Activator Inhibitor Type 1 Up-Regulation Is Associated with Skeletal Muscle Atrophy and Associated Fibrosis
- (2009) Jasmin Naderi et al. AMERICAN JOURNAL OF PATHOLOGY
- Differential MMP-2 and MMP-9 Activity and Collagen Distribution in Skeletal Muscle from pacu(Piaractus mesopotamicus)During Juvenile and Adult Growth Phases
- (2009) Aline Cristina Michelin et al. Anatomical Record-Advances in Integrative Anatomy and Evolutionary Biology
- Urokinase-type plasminogen activator increases hepatocyte growth factor activity required for skeletal muscle regeneration
- (2009) T. H. Sisson et al. BLOOD
- A Profibrotic Effect of Plasminogen Activator Inhibitor Type-1 (PAI-1) in the Heart
- (2009) A. K. M. Tarikuz Zaman et al. EXPERIMENTAL BIOLOGY AND MEDICINE
- Diabetic myopathy differs between Ins2Akita+/− and streptozotocin-induced Type 1 diabetic models
- (2009) Matthew P. Krause et al. JOURNAL OF APPLIED PHYSIOLOGY
- Muscle-Specific Adaptations, Impaired Oxidative Capacity and Maintenance of Contractile Function Characterize Diet-Induced Obese Mouse Skeletal Muscle
- (2009) Karin E. Shortreed et al. PLoS One
- Adiponectin is expressed by skeletal muscle fibers and influences muscle phenotype and function
- (2008) Matthew P. Krause et al. AMERICAN JOURNAL OF PHYSIOLOGY-CELL PHYSIOLOGY
- Distinct patterns of MMP-9 and MMP-2 activity in slow and fast twitch skeletal muscle regeneration in vivo
- (2008) Malgorzata Zimowska et al. INTERNATIONAL JOURNAL OF DEVELOPMENTAL BIOLOGY
- Inhibition of matrix metalloproteinases suppresses the migration of skeletal muscle cells
- (2008) Takanori Nishimura et al. JOURNAL OF MUSCLE RESEARCH AND CELL MOTILITY
Publish scientific posters with Peeref
Peeref publishes scientific posters from all research disciplines. Our Diamond Open Access policy means free access to content and no publication fees for authors.
Learn MoreAsk a Question. Answer a Question.
Quickly pose questions to the entire community. Debate answers and get clarity on the most important issues facing researchers.
Get Started