IRAK-M Expression Limits Dendritic Cell Activation and Proinflammatory Cytokine Production in Response to Helicobacter pylori
Published 2013 View Full Article
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Title
IRAK-M Expression Limits Dendritic Cell Activation and Proinflammatory Cytokine Production in Response to Helicobacter pylori
Authors
Keywords
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Journal
PLoS One
Volume 8, Issue 6, Pages e66914
Publisher
Public Library of Science (PLoS)
Online
2013-06-12
DOI
10.1371/journal.pone.0066914
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- B-Cell-Delivered Gene Therapy Induces Functional T Regulatory Cells and Leads to a Loss of Antigen-Specific Effector Cells
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- Interleukin-17 Is a Critical Mediator of Vaccine-Induced Reduction of Helicobacter Infection in the Mouse Model
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- Helicobacter pylori Immune Escape Is Mediated by Dendritic Cell–Induced Treg Skewing and Th17 Suppression in Mice
- (2009) John Y. Kao et al. GASTROENTEROLOGY
- Critical Role of IL-1 Receptor-Associated Kinase-M in Regulating Chemokine-Dependent Deleterious Inflammation in Murine Influenza Pneumonia
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- Vaccination of Mice Against H pylori Induces a Strong Th-17 Response and Immunity That Is Neutrophil Dependent
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- IL-17 is Involved inHelicobacter pylori-Induced Gastric Inflammatory Responses in a Mouse Model
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- No evidence of an association between polymorphisms in the IRAK-M gene and atopic dermatitis in a German cohort
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