Monocyte Chemoattractant Protein-1/CCL2 Produced by Stromal Cells Promotes Lung Metastasis of 4T1 Murine Breast Cancer Cells
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Title
Monocyte Chemoattractant Protein-1/CCL2 Produced by Stromal Cells Promotes Lung Metastasis of 4T1 Murine Breast Cancer Cells
Authors
Keywords
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Journal
PLoS One
Volume 8, Issue 3, Pages e58791
Publisher
Public Library of Science (PLoS)
Online
2013-03-19
DOI
10.1371/journal.pone.0058791
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- A Distinct Macrophage Population Mediates Metastatic Breast Cancer Cell Extravasation, Establishment and Growth
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- (2008) G. T. Stathopoulos et al. JNCI-Journal of the National Cancer Institute
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- (2008) Alberto Mantovani et al. NATURE
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