BCL11B Is Up-Regulated by EWS/FLI and Contributes to the Transformed Phenotype in Ewing Sarcoma
Published 2013 View Full Article
- Home
- Publications
- Publication Search
- Publication Details
Title
BCL11B Is Up-Regulated by EWS/FLI and Contributes to the Transformed Phenotype in Ewing Sarcoma
Authors
Keywords
-
Journal
PLoS One
Volume 8, Issue 3, Pages e59369
Publisher
Public Library of Science (PLoS)
Online
2013-03-20
DOI
10.1371/journal.pone.0059369
References
Ask authors/readers for more resources
Related references
Note: Only part of the references are listed.- A dual function of Bcl11b/Ctip2 in hippocampal neurogenesis
- (2012) Ruth Simon et al. EMBO JOURNAL
- Role of the transcription factor Bcl11b in development and lymphomagenesis
- (2012) Ryo KOMINAMI PROCEEDINGS OF THE JAPAN ACADEMY SERIES B-PHYSICAL AND BIOLOGICAL SCIENCES
- The BCL11B tumor suppressor is mutated across the major molecular subtypes of T-cell acute lymphoblastic leukemia
- (2011) A. Gutierrez et al. BLOOD
- Oncogene Mutation Profiling of Pediatric Solid Tumors Reveals Significant Subsets of Embryonal Rhabdomyosarcoma and Neuroblastoma with Mutated Genes in Growth Signaling Pathways
- (2011) N. Shukla et al. CLINICAL CANCER RESEARCH
- LSD1 cooperates with CTIP2 to promote HIV-1 transcriptional silencing
- (2011) V. Le Douce et al. NUCLEIC ACIDS RESEARCH
- Modeling Initiation of Ewing Sarcoma in Human Neural Crest Cells
- (2011) Cornelia von Levetzow et al. PLoS One
- Genome-Wide Identification of Bcl11b Gene Targets Reveals Role in Brain-Derived Neurotrophic Factor Signaling
- (2011) Bin Tang et al. PLoS One
- EWS-FLI-1 modulates miRNA145 and SOX2 expression to initiate mesenchymal stem cell reprogramming toward Ewing sarcoma cancer stem cells
- (2010) N. Riggi et al. GENES & DEVELOPMENT
- The TLX1 oncogene drives aneuploidy in T cell transformation
- (2010) Kim De Keersmaecker et al. NATURE MEDICINE
- An Early T Cell Lineage Commitment Checkpoint Dependent on the Transcription Factor Bcl11b
- (2010) L. Li et al. SCIENCE
- The EWS/FLI1 oncogenic protein inhibits expression of the Wnt inhibitor DICKKOPF-1 gene and antagonizes β-catenin/TCF-mediated transcription
- (2009) Diego Navarro et al. CARCINOGENESIS
- LSD1 Is a Subunit of the NuRD Complex and Targets the Metastasis Programs in Breast Cancer
- (2009) Yan Wang et al. CELL
- GLI1 Is a Direct Transcriptional Target of EWS-FLI1 Oncoprotein
- (2009) Elspeth Beauchamp et al. JOURNAL OF BIOLOGICAL CHEMISTRY
- MEME SUITE: tools for motif discovery and searching
- (2009) T. L. Bailey et al. NUCLEIC ACIDS RESEARCH
- p21WAF1 gene promoter is epigenetically silenced by CTIP2 and SUV39H1
- (2009) T Cherrier et al. ONCOGENE
- A Molecular Function Map of Ewing's Sarcoma
- (2009) Maximilian Kauer et al. PLoS One
- Ctip2/Bcl11b controls ameloblast formation during mammalian odontogenesis
- (2009) O. Golonzhka et al. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA
- Microarray analysis of Ewing’s sarcoma family of tumours reveals characteristic gene expression signatures associated with metastasis and resistance to chemotherapy
- (2008) Karl-Ludwig Schaefer et al. EUROPEAN JOURNAL OF CANCER
- Dual Role of COUP-TF-Interacting Protein 2 in Epidermal Homeostasis and Permeability Barrier Formation
- (2008) Olga Golonzhka et al. JOURNAL OF INVESTIGATIVE DERMATOLOGY
- EWS/FLI Mediates Transcriptional Repression via NKX2.2 during Oncogenic Transformation in Ewing's Sarcoma
- (2008) Leah A. Owen et al. PLoS One
- The Fezf2-Ctip2 genetic pathway regulates the fate choice of subcortical projection neurons in the developing cerebral cortex
- (2008) B. Chen et al. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA
- Microsatellites as EWS/FLI response elements in Ewing's sarcoma
- (2008) K. Gangwal et al. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA
Publish scientific posters with Peeref
Peeref publishes scientific posters from all research disciplines. Our Diamond Open Access policy means free access to content and no publication fees for authors.
Learn MoreAsk a Question. Answer a Question.
Quickly pose questions to the entire community. Debate answers and get clarity on the most important issues facing researchers.
Get Started