Huntingtin’s Function in Axonal Transport Is Conserved in Drosophila melanogaster
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Title
Huntingtin’s Function in Axonal Transport Is Conserved in Drosophila melanogaster
Authors
Keywords
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Journal
PLoS One
Volume 8, Issue 3, Pages e60162
Publisher
Public Library of Science (PLoS)
Online
2013-03-29
DOI
10.1371/journal.pone.0060162
References
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Related references
Note: Only part of the references are listed.- Vesicular Glycolysis Provides On-Board Energy for Fast Axonal Transport
- (2013) Diana Zala et al. CELL
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- High-Content Chemical and RNAi Screens for Suppressors of Neurotoxicity in a Huntington's Disease Model
- (2011) Joost Schulte et al. PLoS One
- Huntingtin Is Required for Mitotic Spindle Orientation and Mammalian Neurogenesis
- (2010) Juliette D. Godin et al. NEURON
- Molecular Mechanisms and Potential Therapeutical Targets in Huntington's Disease
- (2010) Chiara Zuccato et al. PHYSIOLOGICAL REVIEWS
- pARIS-htt: an optimised expression platform to study huntingtin reveals functional domains required for vesicular trafficking
- (2010) Raúl Pardo et al. Molecular Brain
- Inactivation of Drosophila Huntingtin affects long-term adult functioning and the pathogenesis of a Huntington's disease model
- (2009) S. Zhang et al. Disease Models & Mechanisms
- Dynactin regulates bidirectional transport of dense-core vesicles in the axon and dendrites of cultured hippocampal neurons
- (2009) D.M. Kwinter et al. NEUROSCIENCE
- Huntingtin as an essential integrator of intracellular vesicular trafficking
- (2009) Juliane P. Caviston et al. TRENDS IN CELL BIOLOGY
- Huntingtin phosphorylation acts as a molecular switch for anterograde/retrograde transport in neurons
- (2008) Emilie Colin et al. EMBO JOURNAL
- Phosphorylation of mutant huntingtin at S421 restores anterograde and retrograde transport in neurons
- (2008) Diana Zala et al. HUMAN MOLECULAR GENETICS
- Enhanced Sensitivity of Striatal Neurons to Axonal Transport Defects Induced by Mutant Huntingtin
- (2008) L.-S. Her et al. JOURNAL OF NEUROSCIENCE
- Suppression of Neurodegeneration and Increased Neurotransmission Caused by Expanded Full-Length Huntingtin Accumulating in the Cytoplasm
- (2008) Eliana Romero et al. NEURON
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