The microRNA -23b/-27b Cluster Suppresses the Metastatic Phenotype of Castration-Resistant Prostate Cancer Cells
Published 2012 View Full Article
- Home
- Publications
- Publication Search
- Publication Details
Title
The microRNA -23b/-27b Cluster Suppresses the Metastatic Phenotype of Castration-Resistant Prostate Cancer Cells
Authors
Keywords
-
Journal
PLoS One
Volume 7, Issue 12, Pages e52106
Publisher
Public Library of Science (PLoS)
Online
2012-12-27
DOI
10.1371/journal.pone.0052106
References
Ask authors/readers for more resources
Related references
Note: Only part of the references are listed.- Vav3-Rac1 Signaling Regulates Prostate Cancer Metastasis with Elevated Vav3 Expression Correlating with Prostate Cancer Progression and Posttreatment Recurrence
- (2012) K.-T. Lin et al. CANCER RESEARCH
- Regulation of microRNA biogenesis and function
- (2012) Thomas Treiber et al. THROMBOSIS AND HAEMOSTASIS
- MiR-27b targets PPARγ to inhibit growth, tumor progression and the inflammatory response in neuroblastoma cells
- (2011) J-J Lee et al. ONCOGENE
- Diagnostic and prognostic signatures from the small non-coding RNA transcriptome in prostate cancer
- (2011) E S Martens-Uzunova et al. ONCOGENE
- The altered expression of MiR-221/-222 and MiR-23b/-27b is associated with the development of human castration resistant prostate cancer
- (2011) Tong Sun et al. PROSTATE
- Genome-wide functional screening of miR-23b as a pleiotropic modulator suppressing cancer metastasis
- (2011) Hanshuo Zhang et al. Nature Communications
- MicroRNA-27b regulates the expression of matrix metalloproteinase 13 in human osteoarthritis chondrocytes
- (2010) Nahid Akhtar et al. ARTHRITIS AND RHEUMATISM
- MicroRNAs and Metastasis: Little RNAs Go a Long Way
- (2010) D. M. Dykxhoorn CANCER RESEARCH
- The Promise of MicroRNA Replacement Therapy
- (2010) A. G. Bader et al. CANCER RESEARCH
- MicroRNAs in malignant progression
- (2010) Li Ma et al. CELL CYCLE
- Activation of Rac1 Is Closely Related to Androgen-Independent Cell Proliferation of Prostate Cancer Cells Bothin Vitroandin Vivo
- (2010) Takashi Kobayashi et al. MOLECULAR ENDOCRINOLOGY
- MicroRNA control of signal transduction
- (2010) Masafumi Inui et al. NATURE REVIEWS MOLECULAR CELL BIOLOGY
- The Role of microRNA-221 and microRNA-222 in Androgen-Independent Prostate Cancer Cell Lines
- (2009) T. Sun et al. CANCER RESEARCH
- MicroRNA-23b mediates urokinase and c-met downmodulation and a decreased migration of human hepatocellular carcinoma cells
- (2009) Alessandro Salvi et al. FEBS Journal
- ST14(Suppression of Tumorigenicity 14) Gene Is a Target for miR-27b, and the Inhibitory Effect of ST14 on Cell Growth Is Independent of miR-27b Regulation
- (2009) Yanfang Wang et al. JOURNAL OF BIOLOGICAL CHEMISTRY
- Changes in the expression of E-cadherin repressors, Snail, Slug, SIP1, and Twist, in the development and progression of ovarian carcinoma: the important role of Snail in ovarian tumorigenesis and progression
- (2009) Junko Yoshida et al. Medical Molecular Morphology
- c-Myc suppression of miR-23a/b enhances mitochondrial glutaminase expression and glutamine metabolism
- (2009) Ping Gao et al. NATURE
- Loss of E-Cadherin Promotes Metastasis via Multiple Downstream Transcriptional Pathways
- (2008) T. T. Onder et al. CANCER RESEARCH
- Genomic Profiling of MicroRNA and Messenger RNA Reveals Deregulated MicroRNA Expression in Prostate Cancer
- (2008) S. Ambs et al. CANCER RESEARCH
- The miR-200 family determines the epithelial phenotype of cancer cells by targeting the E-cadherin repressors ZEB1 and ZEB2
- (2008) S.-M. Park et al. GENES & DEVELOPMENT
- Mammalian Rho GTPases: new insights into their functions from in vivo studies
- (2008) Sarah J. Heasman et al. NATURE REVIEWS MOLECULAR CELL BIOLOGY
- Ligand-Independent Activation of Androgen Receptors by Rho GTPase Signaling in Prostate Cancer
- (2007) Leah S. Lyons et al. MOLECULAR ENDOCRINOLOGY
Find Funding. Review Successful Grants.
Explore over 25,000 new funding opportunities and over 6,000,000 successful grants.
ExploreDiscover Peeref hubs
Discuss science. Find collaborators. Network.
Join a conversation