TAp63γ Demethylation Regulates Protein Stability and Cellular Distribution during Neural Stem Cell Differentiation
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Title
TAp63γ Demethylation Regulates Protein Stability and Cellular Distribution during Neural Stem Cell Differentiation
Authors
Keywords
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Journal
PLoS One
Volume 7, Issue 12, Pages e52417
Publisher
Public Library of Science (PLoS)
Online
2012-12-15
DOI
10.1371/journal.pone.0052417
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- (2010) Massimiliano Agostini et al. BIOCHEMICAL AND BIOPHYSICAL RESEARCH COMMUNICATIONS
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- (2010) Márcia M Aranha et al. BMC GENOMICS
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- (2010) N. Carolina Hernández-Acosta et al. BRAIN RESEARCH
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- TAp73 Acts via the bHLH Hey2 to Promote Long-Term Maintenance of Neural Precursors
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- Ezh2, the histone methyltransferase of PRC2, regulates the balance between self-renewal and differentiation in the cerebral cortex
- (2010) J. D. Pereira et al. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA
- p73 deficiency results in impaired self renewal and premature neuronal differentiation of mouse neural progenitors independently of p53
- (2010) L Gonzalez-Cano et al. Cell Death & Disease
- Regulation of p53 family members by the ubiquitin-like SUMO system
- (2009) Per Stehmeier et al. DNA REPAIR
- Caspases and p53 modulate FOXO3A/Id1 signaling during mouse neural stem cell differentiation
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- (2008) A. Armesilla-Diaz et al. NEUROSCIENCE
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