Opposite Role of Tumor Necrosis Factor Receptors in Dextran Sulfate Sodium-Induced Colitis in Mice

TNFR1-Activated Reactive Oxidative Species Signals Up-Regulate OsteogenicMsx2Programs in Aortic Myofibroblasts

(2012) Chung-Fang Lai et al.   ENDOCRINOLOGY

Colitogenic role of tumour necrosis factor (TNF) receptors in trinitrobenzene sulphonic acid colitis: TNF-R1 ablation does not affect systemic inflammatory response

(2011) Y. Yang et al.   CLINICAL AND EXPERIMENTAL IMMUNOLOGY

Intravenous Injection of Endogenous Microbial Components Abrogates DSS-Induced Colitis

(2011) Beate C. Sydora et al.   DIGESTIVE DISEASES AND SCIENCES

ANCA in the diagnosis of neutrophil-mediated inflammation

(2010) Ivo Lochman et al.   AUTOIMMUNITY REVIEWS

Atm-Deficient Mice Exhibit Increased Sensitivity to Dextran Sulfate Sodium-Induced Colitis Characterized by Elevated DNA Damage and Persistent Immune Activation

(2010) A. M. Westbrook et al.   CANCER RESEARCH

Role of TNF receptors, TNFR1 and TNFR2, in dextran sodium sulfate-induced colitis

(2009) RoseMarie Stillie et al.   INFLAMMATORY BOWEL DISEASES

Tumor necrosis factor-alpha (TNF-α) induces integrin CD11b/CD18 (Mac-1) up-regulation and migration to the CC chemokine CCL3 (MIP-1α) on human neutrophils through defined signalling pathways

(2007) Fabrizio Montecucco et al.   CELLULAR SIGNALLING

The role of chemokines in neutrophil biology

(2007) Yoshiro Kobayashi   Frontiers in Bioscience-Landmark