Splice Variants of NaV1.7 Sodium Channels Have Distinct β Subunit-Dependent Biophysical Properties

Alternative Splicing Modulates Inactivation of Type 1 Voltage-gated Sodium Channels by Toggling an Amino Acid in the First S3-S4 Linker

(2011) Emily V. Fletcher et al.   JOURNAL OF BIOLOGICAL CHEMISTRY

Na+ChannelScn1bGene Regulates Dorsal Root Ganglion Nociceptor Excitabilityin Vivo

(2011) Luis F. Lopez-Santiago et al.   JOURNAL OF BIOLOGICAL CHEMISTRY

Na+ Channel ? Subunits: Overachievers of the Ion Channel Family

(2011) William J. Brackenbury et al.   Frontiers in Pharmacology

Alternative splicing may contribute to time-dependent manifestation of inherited erythromelalgia

(2010) Jin-Sung Choi et al.   BRAIN

Electrophysiology and beyond: Multiple roles of Na+ channel β subunits in development and disease

(2010) Gustavo A. Patino et al.   NEUROSCIENCE LETTERS

A novel Nav1.7 mutation producing carbamazepine-responsive erythromelalgia

(2009) Tanya Z. Fischer et al.   ANNALS OF NEUROLOGY

Differential expression of neuronal voltage-gated sodium channel mRNAs during the development of the rat trigeminal ganglion

(2009) Jonas Thun et al.   BRAIN RESEARCH

Differential expression of exon 5 splice variants of sodium channel α subunit mRNAs in the developing mouse brain

(2009) E.V. Gazina et al.   NEUROSCIENCE

Voltage-Gated Sodium Channels: Therapeutic Targets for Pain

(2009) Sulayman D. Dib-Hajj et al.   PAIN MEDICINE

Sensory neuron voltage-gated sodium channels as analgesic drug targets

(2008) Aliakmal Momin et al.   CURRENT OPINION IN NEUROBIOLOGY