Ablation of the Pro-Apoptotic Protein Bax Protects Mice from Glucocorticoid-Induced Bone Growth Impairment

Effects of short-term glucocorticoid treatment on changes in cartilage matrix degradation and chondrocyte gene expression induced by mechanical injury and inflammatory cytokines

(2011) Yihong CS Lu et al.   ARTHRITIS RESEARCH & THERAPY

“Where, O Death, Is Thy Sting?” A Brief Review of Apoptosis Biology

(2010) Andrew H. Wyllie   MOLECULAR NEUROBIOLOGY

Negative Regulation of Bone Formation by the Transmembrane Wnt Antagonist Kremen-2

(2010) Jochen Schulze et al.   PLoS One

Glucocorticoids increase impairments in learning and memory due to elevated amyloid precursor protein expression and neuronal apoptosis in 12-month old mice

(2009) Wei-Zu Li et al.   EUROPEAN JOURNAL OF PHARMACOLOGY

Catch-up growth after dexamethasone withdrawal occurs in cultured postnatal rat metatarsal bones

(2009) Andrei S Chagin et al.   JOURNAL OF ENDOCRINOLOGY

Bax deficiency in mice increases cartilage production during fracture repair through a mechanism involving increased chondrocyte proliferation without changes in apoptosis

(2008) Charles H. Rundle et al.   BONE

Deregulation of the Mitochondrial Apoptotic Machinery and Development of Molecular Targeted Drugs in Acute Myeloid Leukemia

(2008) G. Poeta et al.   CURRENT CANCER DRUG TARGETS

Bim, Bak, and Bax Regulate Osteoblast Survival

(2008) Min Liang et al.   JOURNAL OF BONE AND MINERAL RESEARCH

Japanese encephalitis virus infection activates caspase-8 and -9 in a FADD-independent and mitochondrion-dependent manner

(2008) C.-H. Tsao et al.   JOURNAL OF GENERAL VIROLOGY

Regulation of bim in glucocorticoid-mediated osteoblast apoptosis

(2007) B. Espina et al.   JOURNAL OF CELLULAR PHYSIOLOGY