Genetic Knock-Down of Hdac3 Does Not Modify Disease-Related Phenotypes in a Mouse Model of Huntington's Disease
Published 2012 View Full Article
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Title
Genetic Knock-Down of Hdac3 Does Not Modify Disease-Related Phenotypes in a Mouse Model of Huntington's Disease
Authors
Keywords
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Journal
PLoS One
Volume 7, Issue 2, Pages e31080
Publisher
Public Library of Science (PLoS)
Online
2012-02-09
DOI
10.1371/journal.pone.0031080
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Note: Only part of the references are listed.- HDAC3 Is a Critical Negative Regulator of Long-Term Memory Formation
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- (2011) Anna Bobrowska et al. PLoS One
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- Chemical Probes Identify a Role for Histone Deacetylase 3 in Friedreich's Ataxia Gene Silencing
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- Genetic Knock-Down of HDAC7 Does Not Ameliorate Disease Pathogenesis in the R6/2 Mouse Model of Huntington's Disease
- (2009) Caroline L. Benn et al. PLoS One
- Formation of Polyglutamine Inclusions in a Wide Range of Non-CNS Tissues in the HdhQ150 Knock-In Mouse Model of Huntington's Disease
- (2009) Hilary Moffitt et al. PLoS One
- Inhibition of specific HDACs and sirtuins suppresses pathogenesis in a Drosophila model of Huntington’s disease
- (2008) Judit Pallos et al. HUMAN MOLECULAR GENETICS
- Zn(II)-dependent histone deacetylase inhibitors: Suberoylanilide hydroxamic acid and trichostatin A
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- Maintenance of cardiac energy metabolism by histone deacetylase 3 in mice
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- The Rpd3/Hda1 family of lysine deacetylases: from bacteria and yeast to mice and men
- (2008) Xiang-Jiao Yang et al. NATURE REVIEWS MOLECULAR CELL BIOLOGY
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