RIP1-Dependent and Independent Effects of Necrostatin-1 in Necrosis and T Cell Activation
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Title
RIP1-Dependent and Independent Effects of Necrostatin-1 in Necrosis and T Cell Activation
Authors
Keywords
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Journal
PLoS One
Volume 6, Issue 8, Pages e23209
Publisher
Public Library of Science (PLoS)
Online
2011-08-11
DOI
10.1371/journal.pone.0023209
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- zVAD-induced necroptosis in L929 cells depends on autocrine production of TNFα mediated by the PKC–MAPKs–AP-1 pathway
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- Virus Inhibition of RIP3-Dependent Necrosis
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- Going up in flames: necrotic cell injury and inflammatory diseases
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- (2010) S. L. Osborn et al. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA
- The molecular regulation of programmed necrotic cell injury
- (2010) David Moquin et al. TRENDS IN BIOCHEMICAL SCIENCES
- Phosphorylation-Driven Assembly of the RIP1-RIP3 Complex Regulates Programmed Necrosis and Virus-Induced Inflammation
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- Identification of RIP1 kinase as a specific cellular target of necrostatins
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- Antigen-mediated T cell expansion regulated by parallel pathways of death
- (2008) I. L. Ch'en et al. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA
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