Control of Alzheimer's Amyloid Beta Toxicity by the High Molecular Weight Immunophilin FKBP52 and Copper Homeostasis in Drosophila

Modulation of glucocorticoid receptor nuclear translocation in neurons by immunophilins FKBP51 and FKBP52: Implications for major depressive disorder

(2009) Erick T. Tatro et al.   BRAIN RESEARCH

Fenton chemistry and oxidative stress mediate the toxicity of the β-amyloid peptide in aDrosophilamodel of Alzheimer’s disease

(2009) Thomas Rival et al.   EUROPEAN JOURNAL OF NEUROSCIENCE

Identifying genes that interact withDrosophilapresenilin and amyloid precursor protein

(2009) Diana L. van de Hoef et al.   GENESIS

Chronic copper exposure exacerbates both amyloid and tau pathology and selectively dysregulates cdk5 in a mouse model of AD

(2009) Masashi Kitazawa et al.   JOURNAL OF NEUROCHEMISTRY

Increasing Cu bioavailability inhibits A  oligomers and tau phosphorylation

(2009) P. J. Crouch et al.   PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA

Intracellular copper deficiency increases amyloid-β secretion by diverse mechanisms

(2008) Michael A. Cater et al.   BIOCHEMICAL JOURNAL

Amyloidogenic metal-binding proteins: new investigative pathways

(2008) Paul Davies et al.   BIOCHEMICAL SOCIETY TRANSACTIONS

Metals in Alzheimer's and Parkinson's Diseases

(2008) Kevin J Barnham et al.   CURRENT OPINION IN CHEMICAL BIOLOGY

Identification of Novel Genes That Modify Phenotypes Induced by Alzheimer's  -Amyloid Overexpression in Drosophila

(2008) W. Cao et al.   GENETICS

FKBP Family Proteins: Immunophilins with Versatile Biological Functions

(2008) Cong Bao Kang et al.   NEUROSIGNALS