A proliferative melanoma cell phenotype is responsive to RAF/MEK inhibition independent of BRAF mutation status
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Title
A proliferative melanoma cell phenotype is responsive to RAF/MEK inhibition independent of BRAF mutation status
Authors
Keywords
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Journal
Pigment Cell & Melanoma Research
Volume 24, Issue 2, Pages 326-333
Publisher
Wiley
Online
2010-12-23
DOI
10.1111/j.1755-148x.2010.00823.x
References
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- Inhibition of Mutated, Activated BRAF in Metastatic Melanoma
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- Integrating BRAF/MEK inhibitors into combination therapy for melanoma
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- Antitumor Efficacy of the Novel RAF Inhibitor GDC-0879 Is Predicted by BRAFV600E Mutational Status and Sustained Extracellular Signal-Regulated Kinase/Mitogen-Activated Protein Kinase Pathway Suppression
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- (2009) Nathalie Dhomen et al. HEMATOLOGY-ONCOLOGY CLINICS OF NORTH AMERICA
- Polyclonality of BRAF Mutations in Acquired Melanocytic Nevi
- (2009) Jingrong Lin et al. JNCI-Journal of the National Cancer Institute
- MEK1 mutations confer resistance to MEK and B-RAF inhibition
- (2009) C. M. Emery et al. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA
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- The Mitogen-Activated Protein/Extracellular Signal-Regulated Kinase Kinase Inhibitor AZD6244 (ARRY-142886) Induces Growth Arrest in Melanoma Cells and Tumor Regression When Combined with Docetaxel
- (2008) N. K. Haass et al. CLINICAL CANCER RESEARCH
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