Journal
PHYSIOLOGY
Volume 28, Issue 4, Pages 216-224Publisher
AMER PHYSIOLOGICAL SOC
DOI: 10.1152/physiol.00011.2013
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Funding
- National Heart, Lung, and Blood Institute [5R01 HL-092141, 5R01 HL-093579, 1U24 HL-094373, 1P20 HL-113452, 5R01 HL-098481]
- Carlyle Fraser Heart Center of Emory University Hospital Midtown
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Exercise promotes cardioprotection in both humans and animals not only by reducing risk factors associated with cardiovascular disease but by reducing myocardial infarction and improving survival following ischemia. This article will define the role that nitric oxide and beta-adrenergic receptors play in mediating the cardioprotective effects of exercise in the setting of ischemia-reperfusion injury.
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